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Consistent Long-Term Therapeutic Efficacy of Human Umbilical Cord Matrix-Derived Mesenchymal Stromal Cells After Myocardial Infarction Despite Individual Differences and Transient Engraftment.
Laundos, Tiago L; Vasques-Nóvoa, Francisco; Gomes, Rita N; Sampaio-Pinto, Vasco; Cruz, Pedro; Cruz, Hélder; Santos, Jorge M; Barcia, Rita N; Pinto-do-Ó, Perpétua; Nascimento, Diana S.
Afiliación
  • Laundos TL; Instituto de Investigação e Inovação em Saúde (i3S), University of Porto, Porto, Portugal.
  • Vasques-Nóvoa F; Instituto Nacional de Engenharia Biomédica (INEB), University of Porto, Porto, Portugal.
  • Gomes RN; Instituto de Ciências Biomédicas Abel Salazar (ICBAS), University of Porto, Porto, Portugal.
  • Sampaio-Pinto V; Instituto de Investigação e Inovação em Saúde (i3S), University of Porto, Porto, Portugal.
  • Cruz P; Instituto Nacional de Engenharia Biomédica (INEB), University of Porto, Porto, Portugal.
  • Cruz H; Cardiovascular RandD Center, Faculty of Medicine of the University of Porto, Porto, Portugal.
  • Santos JM; Department of Internal Medicine, Centro Hospitalar Universitário São João, Porto, Portugal.
  • Barcia RN; Instituto de Investigação e Inovação em Saúde (i3S), University of Porto, Porto, Portugal.
  • Pinto-do-Ó P; Instituto Nacional de Engenharia Biomédica (INEB), University of Porto, Porto, Portugal.
  • Nascimento DS; Instituto de Ciências Biomédicas Abel Salazar (ICBAS), University of Porto, Porto, Portugal.
Front Cell Dev Biol ; 9: 624601, 2021.
Article en En | MEDLINE | ID: mdl-33614654
Human mesenchymal stem cells gather special interest as a universal and feasible add-on therapy for myocardial infarction (MI). In particular, human umbilical cord matrix-derived mesenchymal stromal cells (UCM-MSC) are advantageous since can be easily obtained and display high expansion potential. Using isolation protocols compliant with cell therapy, we previously showed UCM-MSC preserved cardiac function and attenuated remodeling 2 weeks after MI. In this study, UCM-MSC from two umbilical cords, UC-A and UC-B, were transplanted in a murine MI model to investigate consistency and durability of the therapeutic benefits. Both cellular products improved cardiac function and limited adverse cardiac remodeling 12 weeks post-ischemic injury, supporting sustained and long-term beneficial therapeutic effect. Donor associated variability was found in the modulation of cardiac remodeling and activation of the Akt-mTOR-GSK3ß survival pathway. In vitro, the two cell products displayed similar ability to induce the formation of vessel-like structures and comparable transcriptome in normoxia and hypoxia, apart from UCM-MSCs proliferation and expression differences in a small subset of genes associated with MHC Class I. These findings support that UCM-MSC are strong candidates to assist the treatment of MI whilst calling for the discussion on methodologies to characterize and select best performing UCM-MSC before clinical application.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Guideline / Prognostic_studies Idioma: En Revista: Front Cell Dev Biol Año: 2021 Tipo del documento: Article País de afiliación: Portugal Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Guideline / Prognostic_studies Idioma: En Revista: Front Cell Dev Biol Año: 2021 Tipo del documento: Article País de afiliación: Portugal Pais de publicación: Suiza