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A case of Langerhans cell sarcoma on the scalp: Whole-exome sequencing reveals a role of ultraviolet in the pathogenesis.
Katsuragawa, Hiroyuki; Yamada, Yosuke; Ishida, Yoshihiro; Kaku, Yo; Fujimoto, Masakazu; Kataoka, Tatsuki R; Haga, Hironori.
Afiliación
  • Katsuragawa H; Department of Diagnostic Pathology, Kyoto University Hospital, Kyoto, Japan.
  • Yamada Y; Department of Diagnostic Pathology, Kyoto University Hospital, Kyoto, Japan.
  • Ishida Y; Department of Dermatology, Kyoto University Hospital, Kyoto, Japan.
  • Kaku Y; Department of Dermatology, Kyoto University Hospital, Kyoto, Japan.
  • Fujimoto M; Department of Diagnostic Pathology, Kyoto University Hospital, Kyoto, Japan.
  • Kataoka TR; Department of Diagnostic Pathology, Kyoto University Hospital, Kyoto, Japan.
  • Haga H; Department of Diagnostic Pathology, Kyoto University Hospital, Kyoto, Japan.
Pathol Int ; 70(11): 881-887, 2020 Nov.
Article en En | MEDLINE | ID: mdl-33410565
Langerhans cell sarcoma (LCS) is a high-grade neoplasm with overtly malignant cytological features and a Langerhans cell phenotype. The underlying genetic features are poorly understood, and only a few alterations, such as those of the MARK pathway-related genes, CDKN2A and TP53 have been reported. Here we present a 70-year-old male with LCS on the scalp and pulmonary metastasis. The multinodular tumor, 3.0 cm in diameter, consisted of diffusely proliferated pleomorphic cells with numerous mitoses (53/10 HPFs). Immunohistochemically, the tumor cells were positive for CD1a, Langerin and PD-L1, and the Ki-67 labeling index was 50%. These pathological features were consistent with LCS, and were also observed in the metastatic tumor. Whole-exome sequencing revealed that both the primary and metastatic tumors harbored a large number of mutations (>20 mutations/megabase), with deletion of CDKN2A and TP53 mutation, and highlighted that the mutational signature was predominantly characteristic of ultraviolet (UV) exposure (W = 0.828). Our results suggest, for the first time, that DNA damage by UV could accumulate in Langerhans cells and play a role in the pathogenesis of LCS. The high mutational burden and PD-L1 expression in the tumor would provide a rationale for the use of immune checkpoint inhibitors for treatment of unresectable LCS.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Histiocitosis de Células de Langerhans / Sarcoma de Células de Langerhans / Secuenciación del Exoma Tipo de estudio: Diagnostic_studies / Etiology_studies Límite: Aged / Humans / Male Idioma: En Revista: Pathol Int Asunto de la revista: PATOLOGIA Año: 2020 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Australia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Histiocitosis de Células de Langerhans / Sarcoma de Células de Langerhans / Secuenciación del Exoma Tipo de estudio: Diagnostic_studies / Etiology_studies Límite: Aged / Humans / Male Idioma: En Revista: Pathol Int Asunto de la revista: PATOLOGIA Año: 2020 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Australia