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Oncogenic Role of Long Noncoding RNAMALAT1 in Thyroid Cancer Progression through Regulation of the miR-204/IGF2BP2/m6A-MYC Signaling.
Ye, Mao; Dong, Shu; Hou, Haitao; Zhang, Tao; Shen, Minghai.
Afiliación
  • Ye M; Department of General Surgery, The Second Affiliated Hospital of Zhejiang University School of Medicine, Hangzhou 310009, P.R. China.
  • Dong S; Jiangsu Hengrui Medicine Co., Ltd., Lianyungang 200245, P.R. China.
  • Hou H; Department of General Surgery, The Second Affiliated Hospital of Zhejiang University School of Medicine, Hangzhou 310009, P.R. China.
  • Zhang T; Department of Breast and Thyroid Surgery, Tengzhou Central People's Hospital, Tengzhou 277500, P.R. China.
  • Shen M; Department of General Surgery, The Second Affiliated Hospital of Zhejiang University School of Medicine, Hangzhou 310009, P.R. China.
Mol Ther Nucleic Acids ; 23: 1-12, 2021 Mar 05.
Article en En | MEDLINE | ID: mdl-33312756
Accumulating studies highlight the role of long noncoding RNAs (lncRNAs)/microRNAs (miRNAs)/messenger RNAs (mRNAs) as important regulatory networks in various human cancers, including thyroid cancer (TC). This study aimed to investigate a novel regulatory network dependent on lncRNA metastasis-associated lung adenocarcinoma transcript 1 (MALAT1) in relation to TC development. Quantitative reverse transcription polymerase chain reaction (qRT-PCR) and western blot were initially employed to detect the expression of MALAT1, insulin-like growth factor 2 mRNA binding protein 2 (IGF2BP2), and myelocytomatosis (MYC) in TC cells. Interactions among MALAT1, miR-204, and IGF2BP2 were then identified in vitro. The biological processes of proliferation, migration, invasion, and apoptosis were evaluated in vitro via gain- and loss-of-function experiments, followed by in vivo validation using xenograft mice. Our data indicated that MALAT1 and IGF2BP2 were highly expressed, while miR-204 was poorly expressed in TC. IGF2BP2 was verified as a target of miR-204. MALAT1 was found to upregulate IGF2BP2 and enhance MYC expression via m6A modification recognition by competitively binding to miR-204, conferring a stimulatory effect on proliferation, migration, and invasion of TC cells, which was accompanied by weakened tumor growth and cell apoptosis. Altogether, the central findings of our study suggest that MALAT1 contributes to TC progression through the upregulation of IGF2BP2 by binding to miR-204.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Mol Ther Nucleic Acids Año: 2021 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Mol Ther Nucleic Acids Año: 2021 Tipo del documento: Article Pais de publicación: Estados Unidos