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miR-424-3p Contributes to the Malignant Progression and Chemoresistance of Gastric Cancer.
Li, Yongyuan; Liu, Hongjie; Cui, Yu; Chen, Hekai; Cui, Xuejun; Shao, Jianping; Su, Feng; He, Xianghui.
Afiliación
  • Li Y; Department of General Surgery, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
  • Liu H; Department of General Surgery, Tianjin Medical University General Hospital, Tianjin 300052, People's Republic of China.
  • Cui Y; Department of Radiology, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
  • Chen H; Department of General Surgery, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
  • Cui X; Department of General Surgery, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
  • Shao J; Department of General Surgery, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
  • Su F; Department of General Surgery, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
  • He X; Department of General Surgery, The Fifth Central Hospital, Tianjin 300450, People's Republic of China.
Onco Targets Ther ; 13: 12201-12211, 2020.
Article en En | MEDLINE | ID: mdl-33273826
BACKGROUND: Gastric cancer (GC) is one of the most common and lethal malignancies worldwide. Therefore, a better understanding of the mechanism of its malignant progression and chemoresistance will be helpful for the treatment of patients with GC. METHODS: The gene expression profiles downloaded from GEO database and the TargetScan Human were used to identify the key regulation model based on miRNA by bioinformatics analyses. The regulation of miRNA to target was clarified by luciferase assay, qPCR, and Western blotting. Then, the in vitro and in vivo experiments were further conducted by overexpression or knockdown of miRNA and/or target to examine the regulation effects and clarify the mechanism. RESULTS: In the present study, miR-424-3p was identified to be differentially expressed among normal gastric, GC, and chemoresistant GC tissues. Target analysis results indicated that ABCC2, a chemoresistance-related gene, was a regulated target of miR-424-3p. The in vitro and in vivo experiment results further demonstrated that miR-424-3p relied on ABCC2-induced chemoresistance to promote GC proliferation and metastasis. CONCLUSION: Overall, this study revealed that miR-424-3p contributed to the malignant progression and chemoresistance of GC. Thus, miR-424-3p could be a potential target for the treatment of GC.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Onco Targets Ther Año: 2020 Tipo del documento: Article Pais de publicación: Nueva Zelanda

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Onco Targets Ther Año: 2020 Tipo del documento: Article Pais de publicación: Nueva Zelanda