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Chemotherapy induces dynamic immune responses in breast cancers that impact treatment outcome.
Park, Yeon Hee; Lal, Samir; Lee, Jeong Eon; Choi, Yoon-La; Wen, Ji; Ram, Sripad; Ding, Ying; Lee, Soo-Hyeon; Powell, Eric; Lee, Se Kyung; Yu, Jong Han; Ching, Keith A; Nam, Jae-Yong; Kim, Seok Won; Nam, Seok Jin; Kim, Ji-Yeon; Cho, Soo Youn; Park, Seri; Kim, Jinho; Hwang, Soohyn; Kim, Yu Jin; Bonato, Vinicius; Fernandez, Diane; Deng, Shibing; Wang, Shuoguo; Shin, Hyuntae; Kang, Eun-Suk; Park, Woong-Yang; Rejto, Paul A; Bienkowska, Jadwiga; Kan, Zhengyan.
Afiliación
  • Park YH; Samsung Medical Center, Seoul, Korea. yhparkhmo@skku.edu.
  • Lal S; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Lee JE; Samsung Medical Center, Seoul, Korea.
  • Choi YL; Samsung Medical Center, Seoul, Korea.
  • Wen J; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Ram S; Drug Safety R&D, Pfizer, San Diego, CA, USA.
  • Ding Y; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Lee SH; Pfizer Oncology, Seoul, Korea.
  • Powell E; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Lee SK; Samsung Medical Center, Seoul, Korea.
  • Yu JH; Samsung Medical Center, Seoul, Korea.
  • Ching KA; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Nam JY; Samsung Medical Center, Seoul, Korea.
  • Kim SW; Samsung Medical Center, Seoul, Korea.
  • Nam SJ; Samsung Medical Center, Seoul, Korea.
  • Kim JY; Samsung Medical Center, Seoul, Korea.
  • Cho SY; Samsung Medical Center, Seoul, Korea.
  • Park S; Samsung Medical Center, Seoul, Korea.
  • Kim J; Samsung Genome Institute, Samsung Medical Center, Seoul, Korea.
  • Hwang S; Samsung Medical Center, Seoul, Korea.
  • Kim YJ; Samsung Medical Center, Seoul, Korea.
  • Bonato V; Biostatistics, Pfizer, San Diego, CA, USA.
  • Fernandez D; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Deng S; Biostatistics, Pfizer, San Diego, CA, USA.
  • Wang S; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Shin H; Samsung Genome Institute, Samsung Medical Center, Seoul, Korea.
  • Kang ES; Samsung Medical Center, Seoul, Korea.
  • Park WY; Samsung Genome Institute, Samsung Medical Center, Seoul, Korea.
  • Rejto PA; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Bienkowska J; Oncology Research & Development, Pfizer, San Diego, CA, USA.
  • Kan Z; Oncology Research & Development, Pfizer, San Diego, CA, USA. zhengyan.kan@pfizer.com.
Nat Commun ; 11(1): 6175, 2020 12 02.
Article en En | MEDLINE | ID: mdl-33268821
To elucidate the effects of neoadjuvant chemotherapy (NAC), we conduct whole transcriptome profiling coupled with histopathology analyses of a longitudinal breast cancer cohort of 146 patients including 110 pairs of serial tumor biopsies collected before treatment, after the first cycle of treatment and at the time of surgery. Here, we show that cytotoxic chemotherapies induce dynamic changes in the tumor immune microenvironment that vary by subtype and pathologic response. Just one cycle of treatment induces an immune stimulatory microenvironment harboring more tumor infiltrating lymphocytes (TILs) and up-regulation of inflammatory signatures predictive of response to anti-PD1 therapies while residual tumors are immune suppressed at end-of-treatment compared to the baseline. Increases in TILs and CD8+ T cell proportions in response to NAC are independently associated with pathologic complete response. Further, on-treatment immune response is more predictive of treatment outcome than immune features in paired baseline samples although these are strongly correlated.
Asunto(s)
Antígeno B7-H1/genética; Neoplasias de la Mama/tratamiento farmacológico; Carcinoma Ductal de Mama/tratamiento farmacológico; Regulación Neoplásica de la Expresión Génica; Linfocitos Infiltrantes de Tumor/efectos de los fármacos; Terapia Neoadyuvante/métodos; Antraciclinas/uso terapéutico; Antígeno B7-H1/antagonistas & inhibidores; Antígeno B7-H1/inmunología; Neoplasias de la Mama/genética; Neoplasias de la Mama/inmunología; Neoplasias de la Mama/mortalidad; Linfocitos T CD8-positivos/efectos de los fármacos; Linfocitos T CD8-positivos/inmunología; Linfocitos T CD8-positivos/patología; Carcinoma Ductal de Mama/genética; Carcinoma Ductal de Mama/inmunología; Carcinoma Ductal de Mama/mortalidad; Proteínas de Ciclo Celular/genética; Proteínas de Ciclo Celular/inmunología; Ciclofosfamida/uso terapéutico; Supervivencia sin Enfermedad; Docetaxel/uso terapéutico; Receptor alfa de Estrógeno/genética; Receptor alfa de Estrógeno/inmunología; Femenino; Perfilación de la Expresión Génica; Humanos; Inmunidad Innata; Factores Reguladores del Interferón/genética; Factores Reguladores del Interferón/inmunología; Estudios Longitudinales; Linfocitos Infiltrantes de Tumor/inmunología; Linfocitos Infiltrantes de Tumor/patología; Neoplasia Residual; Receptor ErbB-2/genética; Receptor ErbB-2/inmunología; Trastuzumab/uso terapéutico; Resultado del Tratamiento; Microambiente Tumoral/efectos de los fármacos; Microambiente Tumoral/genética; Microambiente Tumoral/inmunología

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Regulación Neoplásica de la Expresión Génica / Linfocitos Infiltrantes de Tumor / Carcinoma Ductal de Mama / Terapia Neoadyuvante / Antígeno B7-H1 Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2020 Tipo del documento: Article Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Regulación Neoplásica de la Expresión Génica / Linfocitos Infiltrantes de Tumor / Carcinoma Ductal de Mama / Terapia Neoadyuvante / Antígeno B7-H1 Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2020 Tipo del documento: Article Pais de publicación: Reino Unido