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Different response of embryos originating from control and obese mice to insulin in vitro.
KSiNanovÁ, Martina; KovaRÍkovÁ, Veronika; SefCÍkovÁ, Zuzana; SpirkovÁ, Alexandra; CikoS, Stefan; Pisko, Jozef; Fabian, Dusan.
Afiliación
  • KSiNanovÁ M; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
  • KovaRÍkovÁ V; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
  • SefCÍkovÁ Z; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
  • SpirkovÁ A; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
  • CikoS S; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
  • Pisko J; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
  • Fabian D; Institute of Animal Physiology, Centre of Biosciences, Slovak Academy of Sciences, 040 01 Kosice, Slovak Republic.
J Reprod Dev ; 67(1): 25-34, 2021 Feb 15.
Article en En | MEDLINE | ID: mdl-33250503
The aim of the present work was to investigate the impact of maternal obesity on DNA methylation in ovulated oocytes, and to compare the response of in vitro-developing preimplantation embryos originating from control and obese mice to insulin. An intergenerational, diet-induced obesity model was used to produce outbred mice with an increased body weight and body fat. Two-cell and eight-cell embryos recovered from obese and control mice were cultured in a medium supplemented with 1 or 10 ng/ml insulin until blastocyst formation. In the derived blastocysts, cell proliferation, differentiation, and death rates were determined. The results of immunochemical visualization of 5-methylcytosine indicated a slightly higher DNA methylation in ovulated metaphase II oocytes recovered from obese females; however, the difference between groups did not reach statistical significance. Expanded blastocysts developed from embryos provided by control dams showed increased mean cell numbers (two and eight-cell embryos exposed to 10 ng/ml), an increased inner-cell-mass/trophectoderm ratio (two-cell embryos exposed to 1 ng/ml and eight-cell embryos exposed to 10 ng/ml), and a reduced level of apoptosis (two and eight-cell embryos exposed to 10 ng/ml). In contrast, embryos originating from obese mice were significantly less sensitive to insulin; indeed, no difference was recorded in any tested variable between the embryos exposed to insulin and those cultured in insulin-free medium. Real-time RT-PCR analysis showed a significant increase in the amount of insulin receptor transcripts in blastocysts recovered from obese dams. These results suggest that maternal obesity might modulate the mitogenic and antiapoptotic responses of preimplantation embryos to insulin.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Desarrollo Embrionario / Insulina / Obesidad Tipo de estudio: Prognostic_studies Límite: Animals / Pregnancy Idioma: En Revista: J Reprod Dev Asunto de la revista: MEDICINA REPRODUTIVA Año: 2021 Tipo del documento: Article Pais de publicación: Japón

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Desarrollo Embrionario / Insulina / Obesidad Tipo de estudio: Prognostic_studies Límite: Animals / Pregnancy Idioma: En Revista: J Reprod Dev Asunto de la revista: MEDICINA REPRODUTIVA Año: 2021 Tipo del documento: Article Pais de publicación: Japón