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Natural variation in the sequestosome-related gene, sqst-5, underlies zinc homeostasis in Caenorhabditis elegans.
Evans, Kathryn S; Zdraljevic, Stefan; Stevens, Lewis; Collins, Kimberly; Tanny, Robyn E; Andersen, Erik C.
Afiliación
  • Evans KS; Molecular Biosciences, Northwestern University, Evanston, Illinois, United States of America.
  • Zdraljevic S; Interdisciplinary Biological Sciences Program, Northwestern University, Evanston, Illinois, United States of America.
  • Stevens L; Molecular Biosciences, Northwestern University, Evanston, Illinois, United States of America.
  • Collins K; Interdisciplinary Biological Sciences Program, Northwestern University, Evanston, Illinois, United States of America.
  • Tanny RE; Molecular Biosciences, Northwestern University, Evanston, Illinois, United States of America.
  • Andersen EC; Molecular Biosciences, Northwestern University, Evanston, Illinois, United States of America.
PLoS Genet ; 16(11): e1008986, 2020 11.
Article en En | MEDLINE | ID: mdl-33175833
Zinc is an essential trace element that acts as a co-factor for many enzymes and transcription factors required for cellular growth and development. Altering intracellular zinc levels can produce dramatic effects ranging from cell proliferation to cell death. To avoid such fates, cells have evolved mechanisms to handle both an excess and a deficiency of zinc. Zinc homeostasis is largely maintained via zinc transporters, permeable channels, and other zinc-binding proteins. Variation in these proteins might affect their ability to interact with zinc, leading to either increased sensitivity or resistance to natural zinc fluctuations in the environment. We can leverage the power of the roundworm nematode Caenorhabditis elegans as a tractable metazoan model for quantitative genetics to identify genes that could underlie variation in responses to zinc. We found that the laboratory-adapted strain (N2) is resistant and a natural isolate from Hawaii (CB4856) is sensitive to micromolar amounts of exogenous zinc supplementation. Using a panel of recombinant inbred lines, we identified two large-effect quantitative trait loci (QTL) on the left arm of chromosome III and the center of chromosome V that are associated with zinc responses. We validated and refined both QTL using near-isogenic lines (NILs) and identified a naturally occurring deletion in sqst-5, a sequestosome-related gene, that is associated with resistance to high exogenous zinc. We found that this deletion is relatively common across strains within the species and that variation in sqst-5 is associated with zinc resistance. Our results offer a possible mechanism for how organisms can respond to naturally high levels of zinc in the environment and how zinc homeostasis varies among individuals.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Zinc / Proteínas de Caenorhabditis elegans / Proteínas Adaptadoras Transductoras de Señales Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: PLoS Genet Asunto de la revista: GENETICA Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Zinc / Proteínas de Caenorhabditis elegans / Proteínas Adaptadoras Transductoras de Señales Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: PLoS Genet Asunto de la revista: GENETICA Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos