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Design and synthesis of 3-(4-pyridyl)-5-(4-sulfamido-phenyl)-1,2,4-oxadiazole derivatives as novel GSK-3ß inhibitors and evaluation of their potential as multifunctional anti-Alzheimer agents.
Wang, Min; Liu, Tongtong; Chen, Shiming; Wu, Mingfei; Han, Jianfei; Li, Zeng.
Afiliación
  • Wang M; The Key Laboratory of Major Autoimmune Diseases, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University; The Key Llaboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, 230032, China.
  • Liu T; The Key Laboratory of Major Autoimmune Diseases, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University; The Key Llaboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, 230032, China.
  • Chen S; The Key Laboratory of Major Autoimmune Diseases, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University; The Key Llaboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, 230032, China.
  • Wu M; The Key Laboratory of Major Autoimmune Diseases, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University; The Key Llaboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, 230032, China.
  • Han J; The Key Laboratory of Major Autoimmune Diseases, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University; The Key Llaboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, 230032, China.
  • Li Z; The Key Laboratory of Major Autoimmune Diseases, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University; The Key Llaboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, 230032, China. Electronic address: lizeng@ahmu.edu.cn.
Eur J Med Chem ; 209: 112874, 2021 Jan 01.
Article en En | MEDLINE | ID: mdl-33017743
Pleiotropic intervention has prominent advantages for complex pathomechanisms, such as Alzheimer's disease (AD). In this study, a series of novel 3-(4-pyridyl)-5-(4- sulfamido-phenyl)-1,2,4-oxadiazole derivatives were designed and synthesized following the multitarget-directed ligand-based strategy. All compounds were evaluated for glycogen synthase kinase 3ß (GSK-3ß) inhibition and antineuroinflammatory and neuroprotective activities. Given that abnormal glucose metabolism plays an important role in AD occurrence and development, the effects of all compounds on glucose consumption in HepG2 cells was evaluated. Compounds 5e and 10b showed good dual potency in GSK-3ß inhibition (IC50: 5e = 1.52 µM, 10b = 0.19 µM) and antineuroinflammatory potency (IC50: 5e = 0.47 ± 0.64 µM, 10b = 6.94 ± 2.33 µM). The effect of compound 10b on glucose consumption was higher than that of positive drug metformin. These compounds exerted a certain neuroprotective effect. Compound 10b dramatically reduced Aß-induced Tau hyperphosphorylation, thus inhibiting GSK-3ß at the cellular level. Notably, compounds 5e and 10b exhibited good inhibitory effects on the formation of intracellular reactive oxygen species (ROS). Moreover, these compounds displayed proper blood-brain barrier permeability and lacked neurotoxicity up to 50 µM concentration. Finally, in vivo experiments revealed that compound 10b improved cognitive impairment in scopolamine-induced mouse models. Results indicated that compound 10b deserves further study as a multifunctional lead compound.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Oxadiazoles / Inhibidores Enzimáticos / Enfermedad de Alzheimer / Glucógeno Sintasa Quinasa 3 beta Límite: Animals / Humans / Male Idioma: En Revista: Eur J Med Chem Año: 2021 Tipo del documento: Article País de afiliación: China Pais de publicación: Francia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Oxadiazoles / Inhibidores Enzimáticos / Enfermedad de Alzheimer / Glucógeno Sintasa Quinasa 3 beta Límite: Animals / Humans / Male Idioma: En Revista: Eur J Med Chem Año: 2021 Tipo del documento: Article País de afiliación: China Pais de publicación: Francia