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Effects of high-dose uric acid on cellular proteome, intracellular ATP, tissue repairing capability and calcium oxalate crystal-binding capability of renal tubular cells: Implications to hyperuricosuria-induced kidney stone disease.
Sutthimethakorn, Suchitra; Thongboonkerd, Visith.
Afiliación
  • Sutthimethakorn S; Medical Proteomics Unit, Office for Research and Development, Faculty of Medicine Siriraj Hospital, Mahidol University, Bangkok, Thailand; Graduate Program in Molecular Medicine, Faculty of Science, Mahidol University, Bangkok, Thailand.
  • Thongboonkerd V; Medical Proteomics Unit, Office for Research and Development, Faculty of Medicine Siriraj Hospital, Mahidol University, Bangkok, Thailand; Center for Research in Complex Systems Science, Mahidol University, Bangkok, Thailand. Electronic address: thongboonkerd@dr.com.
Chem Biol Interact ; 331: 109270, 2020 Nov 01.
Article en En | MEDLINE | ID: mdl-32991862
Hyperuricosuria is associated with kidney stone disease, especially uric acid (UA) and calcium oxalate (CaOx) types. Nevertheless, detailed mechanisms of hyperuricosuria-induced kidney stone formation remained unclear. This study examined changes in cellular proteome and function of renal tubular cells after treatment with high-dose UA for 48-h. Quantitative proteomics using 2-DE followed by nanoLC-ESI-ETD MS/MS tandem mass spectrometry revealed significant changes in levels of 22 proteins in the UA-treated cells. These proteomic data could be confirmed by Western blotting. Functional assays revealed an increase in intracellular ATP level and enhancement of tissue repairing capability in the UA-treated cells. Interestingly, levels of HSP70 and HSP90 (the known receptors for CaOx crystals) were increased in apical membranes of the UA-treated cells. CaOx crystal-cell adhesion assay revealed significant increase in CaOx-binding capability of the UA-treated cells, whereas neutralization of the surface HSP70 and/or HSP90 using their specific monoclonal antibodies caused significant reduction in such binding capability. These findings highlighted changes in renal tubular cells in response to high-dose UA that may, at least in part, explain the pathogenic mechanisms of hyperuricosuria-induced mixed kidney stone disease.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ácido Úrico / Oxalato de Calcio / Adenosina Trifosfato / Proteoma Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: Chem Biol Interact Año: 2020 Tipo del documento: Article País de afiliación: Tailandia Pais de publicación: Irlanda

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ácido Úrico / Oxalato de Calcio / Adenosina Trifosfato / Proteoma Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: Chem Biol Interact Año: 2020 Tipo del documento: Article País de afiliación: Tailandia Pais de publicación: Irlanda