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Transcriptomic analysis by RNA sequencing characterises malignant progression of canine insulinoma from normal tissue to metastatic disease.
Capodanno, Y; Buishand, F O; Pang, L Y; Kirpensteijn, J; Mol, J A; Elders, R; Argyle, D J.
Afiliación
  • Capodanno Y; Laboratory of Fundamental Oncology, National Cancer Research Institute, Tokyo, 103-0045, Japan. ycapodan@ncc.go.jp.
  • Buishand FO; Royal (Dick) School of Veterinary Studies and The Roslin Institute, University of Edinburgh, Edinburgh, EH25 9RG, UK. ycapodan@ncc.go.jp.
  • Pang LY; Royal (Dick) School of Veterinary Studies and The Roslin Institute, University of Edinburgh, Edinburgh, EH25 9RG, UK.
  • Kirpensteijn J; Department of Clinical Sciences of Companion Animals, Faculty of Veterinary Medicine, Utrecht University, Utrecht, The Netherlands.
  • Mol JA; Royal (Dick) School of Veterinary Studies and The Roslin Institute, University of Edinburgh, Edinburgh, EH25 9RG, UK.
  • Elders R; Department of Clinical Sciences of Companion Animals, Faculty of Veterinary Medicine, Utrecht University, Utrecht, The Netherlands.
  • Argyle DJ; Hill's Pet Nutrition, Topeka, KS, USA.
Sci Rep ; 10(1): 11581, 2020 07 14.
Article en En | MEDLINE | ID: mdl-32665562
Insulinomas (INS) are the most common human and canine functioning pancreatic neuroendocrine tumours. The long-term prognosis for malignant INS is poor, because micrometastases are frequently missed during surgery. As human and canine malignant INS share clinical and histopathological features, dogs have been proposed as models for INS research. Using RNA-sequencing, we conducted a pilot study to better understand the underlying molecular mechanisms of canine INS. Normal canine pancreas and lymph node control tissues were compared with primary INS and INS-metastatic lymph nodes, revealing more than 3,000 genes differentially expressed in normal pancreas compared to primary INS. Only 164 genes were differentially expressed between primary INS and INS-metastatic lymph nodes. Hierarchical clustering analysis demonstrated similar genetic profiles in normal pancreas and early clinical stage primary INS, whereas late clinical stage primary INS resembled the genetic profile of INS-metastatic lymph nodes. These findings suggest that markers of malignant behaviour could be identified at the primary site of the disease. Finally, using the REACTOME pathways database, we revealed that an active collagen metabolism, extracellular matrix remodelling, beta-cell differentiation and non-beta-cell trans-differentiation might cause disease progression and hyperinsulinism in INS, identifying major pathways worthy of future research in this currently poorly controlled disease.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedades de los Perros / Transcriptoma / Insulinoma / Proteínas de Neoplasias Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Sci Rep Año: 2020 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedades de los Perros / Transcriptoma / Insulinoma / Proteínas de Neoplasias Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Sci Rep Año: 2020 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Reino Unido