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Haploinsufficiency of the FOXA2 associated with a complex clinical phenotype.
Mohammed, Idris; Al-Khawaga, Sara; Bohanna, David; Shabani, Abdusamea; Khan, Faiyaz; Love, Donald R; Nawaz, Zafar; Hussain, Khalid.
Afiliación
  • Mohammed I; College of Health & Life Sciences, Hamad Bin Khalifa University, Doha, Qatar.
  • Al-Khawaga S; Division of Endocrinology, Department of Pediatric Medicine, Sidra Medicine, Doha, Qatar.
  • Bohanna D; College of Health & Life Sciences, Hamad Bin Khalifa University, Doha, Qatar.
  • Shabani A; Division of Endocrinology, Department of Pediatric Medicine, Sidra Medicine, Doha, Qatar.
  • Khan F; Department of Radiology, Sidra Medicine, Doha, Qatar.
  • Love DR; Division of Pathology Genetics, Department of Pathology, Sidra Medicine, Doha, Qatar.
  • Nawaz Z; Division of Endocrinology, Department of Pediatric Medicine, Sidra Medicine, Doha, Qatar.
  • Hussain K; Department of Radiology, Sidra Medicine, Doha, Qatar.
Mol Genet Genomic Med ; 8(6): e1086, 2020 06.
Article en En | MEDLINE | ID: mdl-32277595
BACKGROUND: There are few reports describing the proximal deletions of the short arm of chromosome 20, making it difficult to predict the likely consequences of these deletions. Most previously reported cases have described the association of 20p11.2 deletions with Alagille syndrome, while there are others that include phenotypes such as panhypopituitarism, craniofacial dysmorphism, polysplenia, autism, and Hirschsprung disease. METHODS: Molecular karyotyping, cytogenetics, and DNA sequencing were undertaken in a child to study the genetic basis of a complex phenotype consisting of craniofacial dysmorphism, ocular abnormalities, ectopic inguinal testes, polysplenia, growth hormone deficiency, central hypothyroidism, and gastrointestinal system anomalies. RESULTS: We report the smallest described de novo proximal 20p11.2 deletion, which deletes only the FOXA2 leading to the above complex phenotype. CONCLUSIONS: Haploinsufficiency of the FOXA2 only gene is associated with a multisystem disorder.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fenotipo / Trastorno Autístico / Anomalías Múltiples / Factor Nuclear 3-beta del Hepatocito / Haploinsuficiencia / Hipotiroidismo Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Child / Humans / Male Idioma: En Revista: Mol Genet Genomic Med Año: 2020 Tipo del documento: Article País de afiliación: Qatar Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fenotipo / Trastorno Autístico / Anomalías Múltiples / Factor Nuclear 3-beta del Hepatocito / Haploinsuficiencia / Hipotiroidismo Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Child / Humans / Male Idioma: En Revista: Mol Genet Genomic Med Año: 2020 Tipo del documento: Article País de afiliación: Qatar Pais de publicación: Estados Unidos