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[Effect of Dilong on expression of fibrogenic factors TGF-ß1 and α-SMA in lung tissue of mice with pulmonary fibrosis].
Wang, Hui-Hui; Meng, Yan-Li; Yang, Zhi-Min; Wang, Xiao-Xi; Xu, Hui-Xing; Wang, Wei-Ming.
Afiliación
  • Wang HH; Heilongjiang Academy of Chinese Medicine Sciences Harbin 150036,China.
  • Meng YL; Heilongjiang Academy of Chinese Medicine Sciences Harbin 150036,China.
  • Yang ZM; Heilongjiang Academy of Chinese Medicine Sciences Harbin 150036,China.
  • Wang XX; Heilongjiang Academy of Chinese Medicine Sciences Harbin 150036,China.
  • Xu HX; Heilongjiang Academy of Chinese Medicine Sciences Harbin 150036,China.
  • Wang WM; Heilongjiang Academy of Chinese Medicine Sciences Harbin 150036,China.
Zhongguo Zhong Yao Za Zhi ; 44(24): 5473-5478, 2019 Dec.
Article en Zh | MEDLINE | ID: mdl-32237397
The aim of this paper was to investigate the effect of Dilong( geosaurus) on the expressions of fibrotic factors TGF-ß1 and α-SMA in bleomycin-induced pulmonary fibrosis mice. The binding ability of Dilong to fibrotic factor TGF-ß1 was initially detected by Biacore technology and verified by in vivo pharmacodynamics. A total of 60 SPF C57 mice were randomly divided into 6 groups. Except the blank group( injecting 0. 08 m L·kg-1 sodium chloride in the trachea),the other five groups were given bleomycin( 4 mg·kg-1) to replicate the pulmonary fibrosis model. After 14 days of drug treatment,the expressions of TGF-ß1 and α-SMA were detected by Masson staining,immunohistochemistry and RT-PCR. The results of Biacore experiment showed that the extract of Dilong was well bound to TGF-ß1 protein in vitro,and the binding value reached 619. 3. Compared with the model group,Masson's results showed that cellulose deposition in high-dose,medium-dose and low-dose Dilong groups decreased to varying degrees. RT-PCR results showed that different doses of Dilong could reduce protein and mRNA expressions of TGF-ß1 and α-SMA to a certain extent in a dose-dependent manner. In conclusion,Dilong could delay the process of pulmonary fibrosis by binding to target protein TGF-ß1 and inhibiting the expression of α-SMA.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fibrosis Pulmonar / Actinas / Factor de Crecimiento Transformador beta1 / Medicina Tradicional China Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals Idioma: Zh Revista: Zhongguo Zhong Yao Za Zhi Asunto de la revista: FARMACOLOGIA / TERAPIAS COMPLEMENTARES Año: 2019 Tipo del documento: Article País de afiliación: China Pais de publicación: China

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fibrosis Pulmonar / Actinas / Factor de Crecimiento Transformador beta1 / Medicina Tradicional China Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals Idioma: Zh Revista: Zhongguo Zhong Yao Za Zhi Asunto de la revista: FARMACOLOGIA / TERAPIAS COMPLEMENTARES Año: 2019 Tipo del documento: Article País de afiliación: China Pais de publicación: China