Hereditary causes of primary aldosteronism and other disorders of apparent excess mineralocorticoid activity.

He, Xin; Modi, Zubin; Else, Tobias

He, Xin; Modi, Zubin; Else, Tobias.

Afiliación

He X; Division of Metabolism, Endocrinology, and Diabetes, University of Michigan, Ann Arbor, MI, USA.
Modi Z; Division of Pediatric Nephrology, Department of Pediatrics, University of Michigan, Ann Arbor, MI, USA.
Else T; Susan B. Meister Child Health Evaluation and Research (CHEAR) Center, University of Michigan, Ann Arbor, MI, USA.

Gland Surg ; 9(1): 150-158, 2020 Feb.

Article en En | MEDLINE | ID: mdl-32206607

Palabras clave

11ß-hydroxylase deficiency; 17α-hydroxylase deficiency; Familial aldosteronism; Liddle syndrome; constitutive activation of the mineralocorticoid receptor; pseudohypoaldosteronism type 2 (PHA2); syndrome of apparent mineralocorticoid excess

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Etiology_studies Idioma: En Revista: Gland Surg Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación:

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