Your browser doesn't support javascript.
loading
Genomic characterisation of breast fibroepithelial lesions in an international cohort.
Md Nasir, Nur Diyana; Ng, Cedric Chuan Young; Rajasegaran, Vikneswari; Wong, Suet Far; Liu, Wei; Ng, Gwendolene Xin Pei; Lee, Jing Yi; Guan, Peiyong; Lim, Jing Quan; Thike, Aye Aye; Koh, Valerie Cui Yun; Loke, Benjamin Nathanael; Chang, Kenneth Tou En; Gudi, Mihir Ananta; Lian, Derrick Wen Quan; Madhukumar, Preetha; Tan, Benita Kiat Tee; Tan, Veronique Kiak Mien; Wong, Chow Yin; Yong, Wei Sean; Ho, Gay Hui; Ong, Kong Wee; Tan, Patrick; Teh, Bin Tean; Tan, Puay Hoon.
Afiliación
  • Md Nasir ND; Department of Anatomical Pathology, Singapore General Hospital, Singapore.
  • Ng CCY; Laboratory of Cancer Epigenome, National Cancer Centre Singapore, Singapore.
  • Rajasegaran V; Cancer and Stem Cell Biology, Duke-NUS Medical School, Singapore.
  • Wong SF; Laboratory of Cancer Epigenome, National Cancer Centre Singapore, Singapore.
  • Liu W; Cancer and Stem Cell Biology, Duke-NUS Medical School, Singapore.
  • Ng GXP; Laboratory of Cancer Epigenome, National Cancer Centre Singapore, Singapore.
  • Lee JY; Laboratory of Cancer Epigenome, National Cancer Centre Singapore, Singapore.
  • Guan P; Laboratory of Cancer Epigenome, National Cancer Centre Singapore, Singapore.
  • Lim JQ; Division of Surgical Oncology, National Cancer Center Singapore, Singapore.
  • Thike AA; Laboratory of Cancer Epigenome, National Cancer Centre Singapore, Singapore.
  • Koh VCY; Cancer and Stem Cell Biology, Duke-NUS Medical School, Singapore.
  • Loke BN; Lymphoma Genomic Translational Laboratory, National Cancer Centre Singapore, Singapore.
  • Chang KTE; Department of Anatomical Pathology, Singapore General Hospital, Singapore.
  • Gudi MA; Department of Anatomical Pathology, Singapore General Hospital, Singapore.
  • Lian DWQ; Department of Anatomical Pathology, Singapore General Hospital, Singapore.
  • Madhukumar P; Yong Loo Lin School of Medicine, National University of Singapore, Singapore.
  • Tan BKT; Department of Pathology and Laboratory Medicine, KK Women's and Children's Hospital, Singapore.
  • Tan VKM; Department of Pathology and Laboratory Medicine, KK Women's and Children's Hospital, Singapore.
  • Wong CY; Department of Pathology and Laboratory Medicine, KK Women's and Children's Hospital, Singapore.
  • Yong WS; Division of Surgical Oncology, National Cancer Center Singapore, Singapore.
  • Ho GH; Department of General Surgery, Singapore General Hospital, Singapore.
  • Ong KW; Division of Surgical Oncology, National Cancer Center Singapore, Singapore.
  • Tan P; Department of Surgery, Sengkang General Hospital, Singapore.
  • Teh BT; Division of Surgical Oncology, National Cancer Center Singapore, Singapore.
  • Tan PH; Department of General Surgery, Singapore General Hospital, Singapore.
J Pathol ; 249(4): 447-460, 2019 12.
Article en En | MEDLINE | ID: mdl-31411343
Fibroepithelial lesions (FELs) are a heterogeneous group of tumours comprising fibroadenomas (FAs) and phyllodes tumours (PTs). Here we used a 16-gene panel that was previously discovered to be implicated in pathogenesis and progression, to characterise a large international cohort of FELs via targeted sequencing. The study comprised 303 (38%) FAs and 493 (62%) PTs which were contributed by the International Fibroepithelial Consortium. There were 659 (83%) Asian and 109 (14%) non-Asian FELs, while the ethnicity of the rest was unknown. Genetic aberrations were significantly associated with increasing grade of PTs, and were detected more in PTs than FAs for MED12, TERT promoter, RARA, FLNA, SETD2, TP53, RB1, EGFR, and IGF1R. Most borderline and malignant PTs possessed ≥ 2 mutations, while there were more cases of FAs with ≤ 1 mutation compared to PTs. FELs with MED12 mutations had significantly higher rates of TERT promoter, RARA, SETD2, EGFR, ERBB4, MAP3K1, and IGF1R aberrations. However, FELs with wild-type MED12 were more likely to express TP53 and PIK3CA mutations. There were no significant differences observed between the mutational profiles of recurrent FAs, FAs with a history of subsequent ipsilateral recurrence or contralateral occurrence, and FAs without a history of subsequent events. We identified recurrent mutations which were more frequent in PTs than FAs, with borderline and malignant PTs harbouring cancer driver gene and multiple mutations. This study affirms the role of a set of genes in FELs, including its potential utility in classification based on mutational profiles. © 2019 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.
Asunto(s)
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Análisis Mutacional de ADN / Biomarcadores de Tumor / Fibroadenoma / Tumor Filoide / Perfilación de la Expresión Génica / Mutación Tipo de estudio: Diagnostic_studies / Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Female / Humans Idioma: En Revista: J Pathol Año: 2019 Tipo del documento: Article País de afiliación: Singapur Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Análisis Mutacional de ADN / Biomarcadores de Tumor / Fibroadenoma / Tumor Filoide / Perfilación de la Expresión Génica / Mutación Tipo de estudio: Diagnostic_studies / Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Female / Humans Idioma: En Revista: J Pathol Año: 2019 Tipo del documento: Article País de afiliación: Singapur Pais de publicación: Reino Unido