Your browser doesn't support javascript.
loading
Autosomal recessive spinocerebellar ataxia SCAR8/ARCA1: First families detected in Spain. / Heredoataxia cerebelosa recesiva ARCA1/SCAR8: primeras familias detectadas en España.
Arias, M; Mir, P; Fernández-Matarrubia, M; Arpa, J; García-Ramos, R; Blanco-Arias, P; Quintans, B; Sobrido, M J.
Afiliación
  • Arias M; Servicio de Neurología, Complexo Hospitalario de Santiago de Compostela, Santiago de Compostela, La Coruña, España. Electronic address: manuel.arias@usc.es.
  • Mir P; Servicio de Neurología, Hospital Virgen del Rocío, Sevilla, España.
  • Fernández-Matarrubia M; Servicio de Neurología, Hospital Clínico San Carlos de, Madrid, España.
  • Arpa J; Servicio de Neurología, Hospital Clínico San Carlos de, Madrid, España.
  • García-Ramos R; Servicio de Neurología, Hospital Clínico San Carlos de, Madrid, España.
  • Blanco-Arias P; Fundación Pública Galega de Medicina Xenómica, Santiago de Compostela, La Coruña, España.
  • Quintans B; Grupo de Neurogenética, Instituto de Investigación Sanitaria de Santiago (IDIS)-Complexo Hospitalario Universitario, Santiago de Compostela, La Coruña, España; Centro de Investigación Biomédica en Red de Enfermedades Raras (CIBERER), Instituto de Salud Carlos III, Madrid, España.
  • Sobrido MJ; Grupo de Neurogenética, Instituto de Investigación Sanitaria de Santiago (IDIS)-Complexo Hospitalario Universitario, Santiago de Compostela, La Coruña, España; Centro de Investigación Biomédica en Red de Enfermedades Raras (CIBERER), Instituto de Salud Carlos III, Madrid, España.
Neurologia (Engl Ed) ; 2019 May 15.
Article en En, Es | MEDLINE | ID: mdl-31103315
INTRODUCTION: Autosomal recessive spinocerebellar ataxia type 8 (ARCA1/SCAR8) is caused by mutations of the SYNE1 gene. The disease was initially described in families from Quebec (Canada) with a phenotype of pure cerebellar syndrome, but in recent years has been reported with a more variable clinical phenotype in other countries. Cases have recently been described of muscular dystrophy, arthrogryposis, and cardiomyopathy due to SYNE1 mutations. OBJECTIVE: To describe clinical and molecular findings from 4 patients (3 men and one woman) diagnosed with ARCA1/SCAR8 from 3 Spanish families from different regions. MATERIAL AND METHODS: We describe the clinical, paraclinical, and genetic results from 4 patients diagnosed with ARCA1/SCAR8 at different Spanish neurology departments. RESULTS: Onset occurred in the third or fourth decade of live in all patients. After 15 years of progression, 3 patients presented pure cerebellar syndrome, similar to the Canadian patients; the fourth patient, with over 30 years' progression, presented vertical gaze palsy, pyramidal signs, and moderate cognitive impairment. In all patients, MRI studies showed cerebellar atrophy. The genetic study revealed distinct pathogenic SYNE1 mutations in each family. CONCLUSIONS: ARCA1/SCAR8 can be found worldwide and may be caused by many distinct mutations in the SYNE1 gene. The disease may manifest with a complex phenotype of varying severity.
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En / Es Revista: Neurologia (Engl Ed) Año: 2019 Tipo del documento: Article Pais de publicación: España

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En / Es Revista: Neurologia (Engl Ed) Año: 2019 Tipo del documento: Article Pais de publicación: España