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Deletion of Cdkn1b in ACI rats leads to increased proliferation and pregnancy-associated changes in the mammary gland due to perturbed systemic endocrine environment.
Ding, Lina; Shunkwiler, Lauren B; Harper, Nicholas W; Zhao, Yang; Hinohara, Kunihiko; Huh, Sung Jin; Ekram, Muhammad B; Guz, Jan; Kern, Michael J; Awgulewitsch, Alexander; Shull, James D; Smits, Bart M G; Polyak, Kornelia.
Afiliación
  • Ding L; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, United States of America.
  • Shunkwiler LB; Department of Medicine, Harvard Medical School, Boston, Massachusetts, United States of America.
  • Harper NW; Department of Pathology and Laboratory Medicine, Medical University of South Carolina, Charleston, South Carolina, United States of America.
  • Zhao Y; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, United States of America.
  • Hinohara K; Department of Pathology and Laboratory Medicine, Medical University of South Carolina, Charleston, South Carolina, United States of America.
  • Huh SJ; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, United States of America.
  • Ekram MB; Department of Medicine, Harvard Medical School, Boston, Massachusetts, United States of America.
  • Guz J; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, United States of America.
  • Kern MJ; Department of Medicine, Harvard Medical School, Boston, Massachusetts, United States of America.
  • Awgulewitsch A; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, United States of America.
  • Shull JD; Department of Medicine, Harvard Medical School, Boston, Massachusetts, United States of America.
  • Smits BMG; Department of Regenerative Medicine and Cell Biology, Transgenic and Gene Function Core, Medical University of South Carolina, Charleston, South Carolina, United States of America.
  • Polyak K; Department of Regenerative Medicine and Cell Biology, Transgenic and Gene Function Core, Medical University of South Carolina, Charleston, South Carolina, United States of America.
PLoS Genet ; 15(3): e1008002, 2019 03.
Article en En | MEDLINE | ID: mdl-30893315
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Inhibidor p27 de las Quinasas Dependientes de la Ciclina Tipo de estudio: Etiology_studies / Risk_factors_studies Límite: Animals / Female / Humans / Pregnancy Idioma: En Revista: PLoS Genet Asunto de la revista: GENETICA Año: 2019 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
Texto completo
Añadir a Mi BVS
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XML
PubMed Links
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Inhibidor p27 de las Quinasas Dependientes de la Ciclina Tipo de estudio: Etiology_studies / Risk_factors_studies Límite: Animals / Female / Humans / Pregnancy Idioma: En Revista: PLoS Genet Asunto de la revista: GENETICA Año: 2019 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos