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Molecular analysis of glycosylphosphatidylinositol anchor deficient aerolysin resistant isolates in gulf war i veterans exposed to depleted uranium.
Nicklas, Janice A; Vacek, Pamela M; Carter, Elizabeth W; McDiarmid, Melissa; Albertini, Richard J.
Afiliación
  • Nicklas JA; Department of Pediatrics, University of Vermont College of Medicine, Burlington, Vermont.
  • Vacek PM; Medical Biostatistics Unit, University of Vermont College of Medicine, Burlington, Vermont.
  • Carter EW; Jeffords Institute for Quality, University of Vermont Medical Center, Burlington, Vermont.
  • McDiarmid M; Occupational Health Program, Department of Medicine, University of Maryland School of Medicine, Baltimore, Maryland.
  • Albertini RJ; U.S. Department of Veterans Affairs, Washington, District of Columbia.
Environ Mol Mutagen ; 60(6): 470-493, 2019 07.
Article en En | MEDLINE | ID: mdl-30848503
During the First Gulf War (1991) over 100 servicemen sustained depleted uranium (DU) exposure through wound contamination, inhalation, and shrapnel. The Department of Veterans Affairs has a surveillance program for these Veterans which has included genotoxicity assays. The frequencies of glycosylphosphatidylinositol anchor (GPIa) negative (aerolysin resistant) cells determined by cloning assays for these Veterans are reported in Albertini RJ et al. (2019: Environ Mol Mutagen). Molecular analyses of the GPIa biosynthesis class A (PIGA) gene was performed on 862 aerolysin-resistant T-lymphocyte recovered isolates. The frequencies of different types of PIGA mutations were compared between high and low DU exposure groups. Additional molecular studies were performed on mutants that produced no PIGA mRNA or with deletions of all or part of the PIGA gene to determine deletion size and breakpoint sequence. One mutant appeared to be the result of a chromothriptic event. A significant percentage (>30%) of the aerolysin resistant isolates, which varied by sample year and Veteran, had wild-type PIGA cDNA (no mutation). As described in Albertini RJ et al. (2019: Environ Mol Mutagen), TCR gene rearrangement analysis of these isolates indicated most arose from multiple T-cell progenitors (hence the inability to find a mutation). It is likely that these isolates were the result of failure of complete selection against nonmutant cells in the cloning assays. Real-time studies of GPIa resistant isolates with no PIGA mutation but with a single TCR gene rearrangement found one clone with a PIGV deletion and several others with decreased levels of GPIa pathway gene mRNAs implying mutation in other GPIa pathway genes. Environ. Mol. Mutagen. 60:470-493, 2019. © 2019 Wiley Periodicals, Inc.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Convulsiones / Toxinas Bacterianas / Exposición Profesional / Uranio / Glicosilfosfatidilinositoles / Proteínas Citotóxicas Formadoras de Poros / Mutágenos Límite: Humans País/Región como asunto: America do norte Idioma: En Revista: Environ Mol Mutagen Asunto de la revista: BIOLOGIA MOLECULAR / SAUDE AMBIENTAL Año: 2019 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Convulsiones / Toxinas Bacterianas / Exposición Profesional / Uranio / Glicosilfosfatidilinositoles / Proteínas Citotóxicas Formadoras de Poros / Mutágenos Límite: Humans País/Región como asunto: America do norte Idioma: En Revista: Environ Mol Mutagen Asunto de la revista: BIOLOGIA MOLECULAR / SAUDE AMBIENTAL Año: 2019 Tipo del documento: Article Pais de publicación: Estados Unidos