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TRPV4 deletion protects against hypokalemia during systemic K+ deficiency.
Tomilin, Viktor; Mamenko, Mykola; Zaika, Oleg; Wingo, Charles S; Pochynyuk, Oleh.
Afiliación
  • Tomilin V; Department of Integrative Biology and Pharmacology, University of Texas Health Science Center at Houston , Houston, Texas.
  • Mamenko M; Department of Physiology, Medical College of Georgia, Augusta University , Augusta, Georgia.
  • Zaika O; Department of Integrative Biology and Pharmacology, University of Texas Health Science Center at Houston , Houston, Texas.
  • Wingo CS; Division of Nephrology, Hypertension and Transplantation, Department of Medicine, University of Florida , Gainesville, Florida.
  • Pochynyuk O; North Florida/South Georgia Veterans Health System, Gainesville, Florida.
Am J Physiol Renal Physiol ; 316(5): F948-F956, 2019 05 01.
Article en En | MEDLINE | ID: mdl-30838874
Tight regulation of K+ balance is fundamental for normal physiology. Reduced dietary K+ intake, which is common in Western diets, often leads to hypokalemia and associated cardiovascular- and kidney-related pathologies. The distal nephron, and, specifically, the collecting duct (CD), is the major site of controlled K+ reabsorption via H+-K+-ATPase in the state of dietary K+ deficiency. We (Mamenko MV, Boukelmoune N, Tomilin VN, Zaika OL, Jensen VB, O'Neil RG, Pochynyuk OM. Kidney Int 91: 1398-1409, 2017) have previously demonstrated that the transient receptor potential vanilloid type 4 (TRPV4) Ca2+ channel, abundantly expressed in the CD, contributes to renal K+ handling by promoting flow-induced K+ secretion. Here, we investigated a potential role of TRPV4 in controlling H+-K+-ATPase-dependent K+ reabsorption in the CD. Treatment with a K+-deficient diet (<0.01% K+) for 7 days reduced serum K+ levels in wild-type (WT) mice from 4.3 ± 0.2 to 3.3 ± 0.2 mM but not in TRPV4-/- mice (4.3 ± 0.1 and 4.2 ± 0.3 mM, respectively). Furthermore, we detected a significant reduction in 24-h urinary K+ levels in TRPV4-/- compared with WT mice upon switching to K+-deficient diet. TRPV4-/- animals also had significantly more acidic urine on a low-K+ diet, but not on a regular (0.9% K+) or high-K+ (5% K+) diet, which is consistent with increased H+-K+-ATPase activity. Moreover, we detected a greatly accelerated H+-K+-ATPase-dependent intracellular pH extrusion in freshly isolated CDs from TRPV4-/- compared with WT mice fed a K+-deficient diet. Overall, our results demonstrate a novel kaliuretic role of TRPV4 by inhibiting H+-K+-ATPase-dependent K+ reabsorption in the CD. We propose that TRPV4 inhibition could be a novel strategy to manage certain hypokalemic states in clinical settings.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Deficiencia de Potasio / Potasio en la Dieta / Canales Catiónicos TRPV / Reabsorción Renal / Hipopotasemia / Túbulos Renales Colectores Límite: Animals Idioma: En Revista: Am J Physiol Renal Physiol Asunto de la revista: FISIOLOGIA / NEFROLOGIA Año: 2019 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Deficiencia de Potasio / Potasio en la Dieta / Canales Catiónicos TRPV / Reabsorción Renal / Hipopotasemia / Túbulos Renales Colectores Límite: Animals Idioma: En Revista: Am J Physiol Renal Physiol Asunto de la revista: FISIOLOGIA / NEFROLOGIA Año: 2019 Tipo del documento: Article Pais de publicación: Estados Unidos