S1P promotes inflammation-induced tube formation by HLECs via the S1PR1/NF-&#954;B pathway.

Zheng, Zhi; Zeng, Yong-Zhi; Ren, Kun; Zhu, Xiao; Tan, Ying; Li, Yi; Li, Qian; Yi, Guang-Hui

S1P promotes inflammation-induced tube formation by HLECs via the S1PR1/NF-κB pathway.

Zheng, Zhi; Zeng, Yong-Zhi; Ren, Kun; Zhu, Xiao; Tan, Ying; Li, Yi; Li, Qian; Yi, Guang-Hui.

Afiliación

Zheng Z; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Zeng YZ; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Ren K; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Zhu X; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Tan Y; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Li Y; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Li Q; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China.
Yi GH; Institute of Cardiovascular Disease, Key Laboratory for Atherosclerology of Hunan Province, University of South China, 28 W Changsheng Road, Hengyang, 421001, Hunan, China. Electronic address: ghyi6108@163.com.

Int Immunopharmacol ; 66: 224-235, 2019 Jan.

Article en En | MEDLINE | ID: mdl-30476824

Asunto(s)

Células Endoteliales/fisiología; Inflamación/inmunología; Linfangiogénesis/inmunología; Organogénesis/inmunología; Receptores de Lisoesfingolípidos/metabolismo; Animales; Movimiento Celular; Proliferación Celular; Células Cultivadas; Humanos; Interleucina-1beta/genética; Interleucina-1beta/metabolismo; Lisofosfolípidos/inmunología; Masculino; Ratones; Ratones Endogámicos C57BL; FN-kappa B/metabolismo; Fosforilación; ARN Interferente Pequeño/genética; Transducción de Señal; Esfingosina/análogos & derivados; Esfingosina/inmunología; Receptores de Esfingosina-1-Fosfato; Factor de Necrosis Tumoral alfa/genética; Factor de Necrosis Tumoral alfa/metabolismo

Palabras clave

Inflammation; Interleukin-1ß; Lymphangiogenesis; Sphingosine-1-phosphate; Tumor necrosis factor-α

Texto completo

Añadir a Mi BVS

Imprimir

XML

PubMed Links

Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Organogénesis / Células Endoteliales / Linfangiogénesis / Receptores de Lisoesfingolípidos / Inflamación Límite: Animals / Humans / Male Idioma: En Revista: Int Immunopharmacol Asunto de la revista: ALERGIA E IMUNOLOGIA / FARMACOLOGIA Año: 2019 Tipo del documento: Article País de afiliación: China Pais de publicación: Países Bajos

Texto completo

Añadir a Mi BVS

Imprimir

XML

PubMed Links

Buscar en Google