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Pol µ dGTP mismatch insertion opposite T coupled with ligation reveals promutagenic DNA repair intermediate.
Çaglayan, Melike; Wilson, Samuel H.
Afiliación
  • Çaglayan M; Genome Integrity and Structural Biology Laboratory, National Institutes of Health, National Institute of Environmental Health Sciences, Research Triangle Park, NC, 27709, USA. caglayanm@ufl.edu.
  • Wilson SH; Department of Biochemistry and Molecular Biology, University of Florida, Gainesville, FL, 32610, USA. caglayanm@ufl.edu.
Nat Commun ; 9(1): 4213, 2018 10 11.
Article en En | MEDLINE | ID: mdl-30310068
Incorporation of mismatched nucleotides during DNA replication or repair leads to transition or transversion mutations and is considered as a predominant source of base substitution mutagenesis in cancer cells. Watson-Crick like dG:dT base pairing is considered to be an important source of genome instability. Here we show that DNA polymerase (pol) µ insertion of 7,8-dihydro-8'-oxo-dGTP (8-oxodGTP) or deoxyguanosine triphosphate (dGTP) into a model double-strand break DNA repair substrate with template base T results in efficient ligation by DNA ligase. These results indicate that pol µ-mediated dGTP mismatch insertion opposite template base T coupled with ligation could be a feature of mutation prone nonhomologous end joining during double-strand break repair.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Timina / Mutagénesis / Nucleótidos de Desoxiguanina / ADN Polimerasa Dirigida por ADN / Reparación del ADN Límite: Humans Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Timina / Mutagénesis / Nucleótidos de Desoxiguanina / ADN Polimerasa Dirigida por ADN / Reparación del ADN Límite: Humans Idioma: En Revista: Nat Commun Asunto de la revista: BIOLOGIA / CIENCIA Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido