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Antioxidant N-acetylcysteine inhibits maladaptive myocyte autophagy in pressure overload induced cardiac remodeling in rats.
Li, Bao; Sun, Yi; Wang, Jia-Pu; Chi, Rui-Fang; Wang, Ke; Yang, Zi-Jian; Qin, Fu-Zhong; Fan, Bianai.
Afiliación
  • Li B; The Second Hospital of Shanxi Medical University, Taiyuan 030001, Shanxi, PR China; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China.
  • Sun Y; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China.
  • Wang JP; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China.
  • Chi RF; The Second Hospital of Shanxi Medical University, Taiyuan 030001, Shanxi, PR China; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China.
  • Wang K; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China.
  • Yang ZJ; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China.
  • Qin FZ; The Second Hospital of Shanxi Medical University, Taiyuan 030001, Shanxi, PR China; Shanxi Medical University, Taiyuan 030001, Shanxi, PR China. Electronic address: sxcvh13@sina.com.
  • Fan B; Schepens Eye Research Institute, Massachusetts Eye and Ear, Harvard Medical School Affiliate, Boston, MA 02114, USA.
Eur J Pharmacol ; 839: 47-56, 2018 Nov 15.
Article en En | MEDLINE | ID: mdl-30194941
Increased oxidative stress and myocyte autophagy co-exist in cardiac remodeling. However, it is unclear whether oxidative stress mediates maladaptive myocyte autophagy in pathological ventricular remodeling. In this study, we tested the hypothesis that antioxidants prevent maladaptive myocyte autophagy in pressure overload-induced left ventricular (LV) remodeling. Sprague-Dawley rats underwent abdominal aortic constriction (AAC) or sham operation. The animals were randomized to receive an antioxidant N-acetylcysteine (NAC), an autophagy inhibitor 3-methyladenine (3-MA) or placebo treatment for 2 weeks. We measured LV structure and function by echocardiography and hemodynamics, myocyte autophagy and oxidative stress assessed by 8-hydroxy-2-deoxyguanosine (8-OHdG). AAC rats exhibited increased LV hypertrophy assessed by LV wall thickness and myocyte cross-sectional area. NAC prevented LV hypertrophy in AAC rats. There were no significant differences in LV fractional shortening, end-diastolic dimension and the maximal rate of LV pressure rise among the groups. AAC rats showed an increase in myocardial 8-OHdG that was prevented by NAC. The expression of LC3 II protein, a marker of autophagy, was increased at 2 weeks after AAC. Immunohistochemical scores further confirmed the increase in LC3 expression in AAC rats. The expression of autophagic proteins Beclin1 and Atg12 and ERK activity were also increased in AAC rats. NAC prevented the increases in LC3 II protein, LC3 scores, Beclin1, Atg12 and ERK activity in AAC rats. Inhibition of autophagy by 3-MA prevented LV hypertrophy after pressure overload. These findings suggest that antioxidants may be of value to prevent pressure overload-induced cardiac remodeling through inhibition of maladaptive myocyte autophagy.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Acetilcisteína / Autofagia / Presión Sanguínea / Adaptación Fisiológica / Remodelación Ventricular / Miocitos Cardíacos Límite: Animals Idioma: En Revista: Eur J Pharmacol Año: 2018 Tipo del documento: Article Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Acetilcisteína / Autofagia / Presión Sanguínea / Adaptación Fisiológica / Remodelación Ventricular / Miocitos Cardíacos Límite: Animals Idioma: En Revista: Eur J Pharmacol Año: 2018 Tipo del documento: Article Pais de publicación: Países Bajos