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The chromatin nuclear protein NUPR1L is intrinsically disordered and binds to the same proteins as its paralogue.
Neira, José L; López, María Belén; Sevilla, Paz; Rizzuti, Bruno; Cámara-Artigas, Ana; Vidal, Miguel; Iovanna, Juan L.
Afiliación
  • Neira JL; Instituto de Biología Molecular y Celular, Universidad Miguel Hernández, 03202 Elche, Alicante, Spain jlneira@umh.es.
  • López MB; Instituto de Biocomputación y Física de Sistemas Complejos, Joint Units IQFR-CSIC-BIFI, and GBsC-CSIC-BIFI, Universidad de Zaragoza, 50009 Zaragoza, Spain.
  • Sevilla P; Centre de Recherche en Cancérologie de Marseille (CRCM), INSERM U1068, CNRS UMR 7258, Aix-Marseille Université and Institut Paoli-Calmettes, Parc Scientifique et Technologique de Luminy, 13288 Marseille, France.
  • Rizzuti B; Facultad de Farmacia, Departamento de Química Física II, Universidad Complutense de Madrid, 28040 Madrid, Spain.
  • Cámara-Artigas A; Instituto de Estructura de laMateria, IEM-CSIC, Serrano 121, 28006 Madrid, Spain.
  • Vidal M; CNR-NANOTEC, Licryl-UOS Cosenza and CEMIF.Cal, Department of Physics, University of Calabria, 87036 Rende, Italy.
  • Iovanna JL; Department of Chemistry and Physics, Research Centre CIAIMBITAL, University of Almería- ceiA3, 04120 Almería, Spain.
Biochem J ; 475(14): 2271-2291, 2018 07 26.
Article en En | MEDLINE | ID: mdl-29925531
NUPR1 is a protumoral multifunctional intrinsically disordered protein (IDP), which is activated during the acute phases of pancreatitis. It interacts with other IDPs such as prothymosin α, as well as with folded proteins such as the C-terminal region of RING1-B (C-RING1B) of the Polycomb complex; in all those interactions, residues around Ala33 and Thr68 (the 'hot-spot' region) of NUPR1 intervene. Its paralogue, NUPR1L, is also expressed in response to DNA damage, it is p53-regulated, and its expression down-regulates that of the NUPR1 gene. In this work, we characterized the conformational preferences of isolated NUPR1L and its possible interactions with the same molecular partners of NUPR1. Our results show that NUPR1L was an oligomeric IDP from pH 2.0 to 12.0, as judged by steady-state fluorescence, circular dichroism (CD), dynamic light scattering, 1D 1H-NMR (nuclear magnetic resonance), and as indicated by structural modelling. However, in contrast with NUPR1, there was evidence of local helical- or turn-like structures; these structures were not rigid, as judged by the lack of sigmoidal behaviour in the chemical and thermal denaturation curves obtained by CD and fluorescence. Interestingly enough, NUPR1L interacted with prothymosin α and C-RING1B, and with a similar affinity to that of NUPR1 (in the low micromolar range). Moreover, NUPR1L hetero-associated with NUPR1 with an affinity of 0.4 µM and interacted with the 'hot-spot' region of NUPR1. Thus, we suggest that the regulation of NUPR1 gene by NUPR1L does not only happen at the DNA level, but it could also involve direct interactions with NUPR1 natural partners.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Represoras / Factores de Transcripción con Motivo Hélice-Asa-Hélice Básico / Multimerización de Proteína / Proteínas Intrínsecamente Desordenadas / Proteínas de Neoplasias Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Biochem J Año: 2018 Tipo del documento: Article País de afiliación: España Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Represoras / Factores de Transcripción con Motivo Hélice-Asa-Hélice Básico / Multimerización de Proteína / Proteínas Intrínsecamente Desordenadas / Proteínas de Neoplasias Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Biochem J Año: 2018 Tipo del documento: Article País de afiliación: España Pais de publicación: Reino Unido