Your browser doesn't support javascript.
loading
Arabidopsis inositol phosphate kinases IPK1 and ITPK1 constitute a metabolic pathway in maintaining phosphate homeostasis.
Kuo, Hui-Fen; Hsu, Yu-Ying; Lin, Wei-Chi; Chen, Kai-Yu; Munnik, Teun; Brearley, Charles A; Chiou, Tzyy-Jen.
Afiliación
  • Kuo HF; Agricultural Biotechnology Research Center, Academia Sinica, Taipei, 115, Taiwan.
  • Hsu YY; Agricultural Biotechnology Research Center, Academia Sinica, Taipei, 115, Taiwan.
  • Lin WC; Agricultural Biotechnology Research Center, Academia Sinica, Taipei, 115, Taiwan.
  • Chen KY; Agricultural Biotechnology Research Center, Academia Sinica, Taipei, 115, Taiwan.
  • Munnik T; Swammerdam Institute for Life Sciences, University of Amsterdam, Science Park 904, Amsterdam, 1098XH, The Netherlands.
  • Brearley CA; School of Biological Sciences, University of East Anglia, Norwich Research Park, Norwich, NR4 7TJ, UK.
  • Chiou TJ; Agricultural Biotechnology Research Center, Academia Sinica, Taipei, 115, Taiwan.
Plant J ; 2018 May 19.
Article en En | MEDLINE | ID: mdl-29779236
Emerging studies have suggested that there is a close link between inositol phosphate (InsP) metabolism and cellular phosphate (Pi ) homeostasis in eukaryotes; however, whether a common InsP species is deployed as an evolutionarily conserved metabolic messenger to mediate Pi signaling remains unknown. Here, using genetics and InsP profiling combined with Pi -starvation response (PSR) analysis in Arabidopsis thaliana, we showed that the kinase activity of inositol pentakisphosphate 2-kinase (IPK1), an enzyme required for phytate (inositol hexakisphosphate; InsP6 ) synthesis, is indispensable for maintaining Pi homeostasis under Pi -replete conditions, and inositol 1,3,4-trisphosphate 5/6-kinase 1 (ITPK1) plays an equivalent role. Although both ipk1-1 and itpk1 mutants exhibited decreased levels of InsP6 and diphosphoinositol pentakisphosphate (PP-InsP5 ; InsP7 ), disruption of another ITPK family enzyme, ITPK4, which correspondingly caused depletion of InsP6 and InsP7 , did not display similar Pi -related phenotypes, which precludes these InsP species from being effectors. Notably, the level of d/l-Ins(3,4,5,6)P4 was concurrently elevated in both ipk1-1 and itpk1 mutants, which showed a specific correlation with the misregulated Pi phenotypes. However, the level of d/l-Ins(3,4,5,6)P4 is not responsive to Pi starvation that instead manifests a shoot-specific increase in the InsP7 level. This study demonstrates a more nuanced picture of the intersection of InsP metabolism and Pi homeostasis and PSRs than has previously been elaborated, and additionally establishes intermediate steps to phytate biosynthesis in plant vegetative tissues.
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Plant J Asunto de la revista: BIOLOGIA MOLECULAR / BOTANICA Año: 2018 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Plant J Asunto de la revista: BIOLOGIA MOLECULAR / BOTANICA Año: 2018 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Reino Unido