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Endocrine disrupting activities and immunomodulatory effects in lymphoblastoid cell lines of diclofenac, 4-hydroxydiclofenac and paracetamol.
Klopcic, Ivana; Markovic, Tijana; Mlinaric-Rascan, Irena; Sollner Dolenc, Marija.
Afiliación
  • Klopcic I; Faculty of Pharmacy, University of Ljubljana, Askerceva 7, 1000 Ljubljana, Slovenia.
  • Markovic T; Faculty of Pharmacy, University of Ljubljana, Askerceva 7, 1000 Ljubljana, Slovenia.
  • Mlinaric-Rascan I; Faculty of Pharmacy, University of Ljubljana, Askerceva 7, 1000 Ljubljana, Slovenia.
  • Sollner Dolenc M; Faculty of Pharmacy, University of Ljubljana, Askerceva 7, 1000 Ljubljana, Slovenia. Electronic address: marija.sollner@ffa.uni-lj.si.
Toxicol Lett ; 294: 95-104, 2018 Sep 15.
Article en En | MEDLINE | ID: mdl-29777833
A critical literature review reveals that knowledge of side effects of pharmaceuticals diclofenac and paracetamol is extremely important because of their widespread use and occurrence in the environment. In order to delineate whether these compounds have endocrine activity and influence on the immune system, we assessed the potential endocrine disrupting and immunomodulatory activities of: diclofenac (DIC), its metabolite 4-hydroxydiclofenac (4-HD) and paracetamol (PAR). Herein, we report on their impact on estrogen receptor (ER), androgen receptor (AR), glucocorticoid receptor (GR) and thyroid hormone receptor (TR). The endocrine disrupting effects were assessed in vitro in MDA-kb2 and GH3.TRE-Luc cell lines and by the XenoScreen YES/YAS assay. Moreover, binding affinity to nuclear receptors (GR and AR) was also measured. Immunomodulatory properties of the compounds were evaluated in lymphoblastoid cell lines. All the tested compounds showed endocrine disrupting and immunomodulatory activities. The results revealed that both DIC and its metabolite 4-HD exhibited significant estrogenic, anti-androgenic (in YAS assay), (anti)-androgenic, (anti)-glucocorticoid and anti-thyroid hormonal activities (in luciferase reporter gene assays). DIC showed direct binding to the GR, while its metabolite 4-HD to the GR and AR. Only metabolite 4-HD showed estrogenic, androgenic (in YAS assay) and thyroid-hormonal activities. PAR had anti-androgenic activity and anti-thyroid hormonal activity. PAR displayed GR agonist activity with competition to its receptor and agonistic activity to AR. All of the compounds significantly modulated pro-inflammatory and immunoregulatory cytokine production in lymphoblastoid cell lines and were thus proven immunomodulatory. The study is useful in determining toxicological effects and contributes to the knowledge of possible side effects of diclofenac, its metabolite and paracetamol.
Asunto(s)
Acetaminofén/efectos adversos; Analgésicos no Narcóticos/efectos adversos; Antiinflamatorios no Esteroideos/efectos adversos; Diclofenaco/efectos adversos; Disruptores Endocrinos/efectos adversos; Factores Inmunológicos/efectos adversos; Linfocitos/efectos de los fármacos; Acetaminofén/química; Acetaminofén/metabolismo; Analgésicos no Narcóticos/química; Analgésicos no Narcóticos/metabolismo; Antagonistas de Receptores Androgénicos/efectos adversos; Antagonistas de Receptores Androgénicos/química; Antagonistas de Receptores Androgénicos/metabolismo; Andrógenos/efectos adversos; Andrógenos/química; Andrógenos/metabolismo; Antiinflamatorios no Esteroideos/química; Antiinflamatorios no Esteroideos/metabolismo; Unión Competitiva; Línea Celular; Supervivencia Celular/efectos de los fármacos; Células Cultivadas; Citocinas/agonistas; Citocinas/metabolismo; Diclofenaco/análogos & derivados; Diclofenaco/química; Diclofenaco/metabolismo; Disruptores Endocrinos/química; Disruptores Endocrinos/metabolismo; Estrógenos/efectos adversos; Estrógenos/química; Estrógenos/metabolismo; Genes Reporteros/efectos de los fármacos; Humanos; Factores Inmunológicos/química; Factores Inmunológicos/metabolismo; Linfocitos/citología; Linfocitos/inmunología; Linfocitos/metabolismo; Receptores Androgénicos/química; Receptores Androgénicos/genética; Receptores Androgénicos/metabolismo; Receptores de Estrógenos/química; Receptores de Estrógenos/genética; Receptores de Estrógenos/metabolismo; Receptores de Glucocorticoides/agonistas; Receptores de Glucocorticoides/antagonistas & inhibidores; Receptores de Glucocorticoides/genética; Receptores de Glucocorticoides/metabolismo; Receptores de Hormona Tiroidea/agonistas; Receptores de Hormona Tiroidea/antagonistas & inhibidores; Receptores de Hormona Tiroidea/genética; Receptores de Hormona Tiroidea/metabolismo; Proteínas Recombinantes de Fusión/química; Proteínas Recombinantes de Fusión/metabolismo; Relación Estructura-Actividad
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos / Antiinflamatorios no Esteroideos / Diclofenaco / Analgésicos no Narcóticos / Disruptores Endocrinos / Factores Inmunológicos / Acetaminofén Idioma: En Revista: Toxicol Lett Año: 2018 Tipo del documento: Article País de afiliación: Eslovenia Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos / Antiinflamatorios no Esteroideos / Diclofenaco / Analgésicos no Narcóticos / Disruptores Endocrinos / Factores Inmunológicos / Acetaminofén Idioma: En Revista: Toxicol Lett Año: 2018 Tipo del documento: Article País de afiliación: Eslovenia Pais de publicación: Países Bajos