Angiotensin II, bombesin and naloxone in tooth pulp and cutaneous nociceptive mechanisms in rabbits.
Physiol Bohemoslov
; 37(3): 281-5, 1988.
Article
en En
| MEDLINE
| ID: mdl-2975796
The effects of angiotensin II (A II), bombesin (B) and naloxone (N) on the amplitude of the late component of the evoked potentials of the cortex (EP) were studied by electrocutaneous (ECS) and tooth pulp (ETS) stimulation. An intraventricular (50 ng/kg) or intravenous (5 micrograms/kg) injections decreased the amplitude of the negative-positive component with a 20-40 ms latency (NP20-40) EP to ETS, but not to ECS. Saralasin (A II antagonist) injected intraventricularly (130 ng/kg) abolished this effect of A II in response to ETS. N intraventricular injections (30 micrograms/kg) increased the amplitude of NP20-40 EP to ETS, but not to ECS. B (intraventricular injection 20 ng/kg decreased the amplitude of NP20-40 EP to ECS, but not to ETS. This suggests there are the specific mechanisms in analgesia induced by A II and B in the pathways activated by ECS and ETS.
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Colección:
01-internacional
Base de datos:
MEDLINE
Asunto principal:
Fenómenos Fisiológicos de la Piel
/
Nociceptores
/
Angiotensina II
/
Bombesina
/
Corteza Cerebral
/
Ventrículos Cerebrales
/
Pulpa Dental
/
Naloxona
Límite:
Animals
Idioma:
En
Revista:
Physiol Bohemoslov
Año:
1988
Tipo del documento:
Article
Pais de publicación:
República Checa