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The inflammatory state of adipose tissue is not affected by the anti-inflammatory response of the A2a-adenosine system and miR-221/PTEN.
Cortés-Garcia, J D; Briones-Espinoza, M J; Vega-Cárdenas, M; Ruíz-Rodríguez, V M; Mendez-Mancilla, A; Gómez-Otero, Arturo E; Vargas Morales, J M; García-Hernández, M H; Portales-Pérez, D P.
Afiliación
  • Cortés-Garcia JD; Laboratory of Immunology and Cellular and Molecular Biology, Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico.
  • Briones-Espinoza MJ; Laboratory of Immunology and Cellular and Molecular Biology, Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico.
  • Vega-Cárdenas M; Laboratory of Immunology and Cellular and Molecular Biology, Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico.
  • Ruíz-Rodríguez VM; Laboratory of Immunology and Cellular and Molecular Biology, Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico.
  • Mendez-Mancilla A; Laboratory of Immunology and Cellular and Molecular Biology, Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico.
  • Gómez-Otero AE; Aesthetic and Corrective Plastic Surgery Clinic, San Luis Potosí, S.L.P., Mexico.
  • Vargas Morales JM; Laboratory of Clinical Analysis "Dr. Pedro Medina de los Santos", Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico.
  • García-Hernández MH; Medical Research Unit-Zacatecas (UIMZ), Instituto Mexicano del Seguro Social, IMSS, Zacatecas City, Mexico.
  • Portales-Pérez DP; Laboratory of Immunology and Cellular and Molecular Biology, Faculty of Chemical Sciences, UASLP, San Luis Potosí, S.L.P., Mexico. Electronic address: dportale@uaslp.mx.
Int J Biochem Cell Biol ; 100: 42-48, 2018 07.
Article en En | MEDLINE | ID: mdl-29729889
Obesity is a chronic inflammatory state with cytokines, adipokines, and miRNAs. The A2a-adenosine system decreases activation and cytokine release in immune cells. MiR-221 is upregulated in carcinogenesis and inflammatory processes, where its targets PTEN and ETS-1, negatively regulates the Akt pathway and induces the release of pro-inflammatory cytokines, respectively. However, the roles of the A2a-adenosine system and miR-221 in adipose tissue are unknown. The aim of this work was to evaluate the A2a-adenosine and miRNA pathways as immune modulators in adipose tissue. We collected aspirate of adipose tissue from patients with BMI < 25 kg/m2 (BMI < 25) and BMI ≥ 25 kg/m2 (BMI ≥ 25) who underwent liposuction; the adipose tissue was digested with collagenase, and then a Ficoll gradient was performed to obtain mononuclear cells from adipose tissue (MCAT). We evaluated the A2a levels by quantitative Retro-transcriptase Polymerase Chain Reaction (RT-qPCR) and flow cytometry and the A2a-adenosine function with a proliferation assay or cytokine levels in the presence or absence of NAD+, activators, and inhibitors of the system. We also analyzed miR-221, ETS-1 and PTEN levels by qRT-PCR. First, we detected that MCAT presented higher basal proliferation than mononuclear cells from peripheral blood; however, activation of the A2a receptor downregulated cell proliferation and cytokine release. Interestingly, while miR-221 was downregulated in MCAT from subjects with BMI ≥ 25 compared to BMI < 25, their targets ETS-1 and PTEN, were increased. In conclusion, the A2a-adenosine system is decreased in MCAT, but it maintains its function; moreover, miR-221 could participate in promoting inflammation in adipose tissue.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Tejido Adiposo / MicroARNs / Receptor de Adenosina A2A / Fosfohidrolasa PTEN Límite: Adult / Female / Humans / Male Idioma: En Revista: Int J Biochem Cell Biol Asunto de la revista: BIOQUIMICA Año: 2018 Tipo del documento: Article País de afiliación: México Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Tejido Adiposo / MicroARNs / Receptor de Adenosina A2A / Fosfohidrolasa PTEN Límite: Adult / Female / Humans / Male Idioma: En Revista: Int J Biochem Cell Biol Asunto de la revista: BIOQUIMICA Año: 2018 Tipo del documento: Article País de afiliación: México Pais de publicación: Países Bajos