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Rlip depletion prevents spontaneous neoplasia in TP53 null mice.
Awasthi, Sanjay; Tompkins, Joshua; Singhal, Jyotsana; Riggs, Arthur D; Yadav, Sushma; Wu, Xiwei; Singh, Sharda; Warden, Charles; Liu, Zheng; Wang, Jinhui; Slavin, Thomas P; Weitzel, Jeffrey N; Yuan, Yate-Ching; Awasthi, Meenakshi; Srivastava, Satish K; Awasthi, Yogesh C; Singhal, Sharad S.
Afiliación
  • Awasthi S; Division of Hematology & Oncology, Department of Internal Medicine, Texas Tech Health Sciences Center, Lubbock, TX 79430-9410; sanjay.awasthi@ttuhsc.edu ariggs@coh.org.
  • Tompkins J; Diabetes and Metabolism Research Institute, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Singhal J; Diabetes and Metabolism Research Institute, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Riggs AD; Diabetes and Metabolism Research Institute, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Yadav S; Diabetes and Metabolism Research Institute, City of Hope Comprehensive Cancer Center, Duarte, CA 91010; sanjay.awasthi@ttuhsc.edu ariggs@coh.org.
  • Wu X; Diabetes and Metabolism Research Institute, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Singh S; Integrative Genomics Core, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Warden C; Division of Hematology & Oncology, Department of Internal Medicine, Texas Tech Health Sciences Center, Lubbock, TX 79430-9410.
  • Liu Z; Integrative Genomics Core, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Wang J; Bioinformatics Core, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Slavin TP; Integrative Genomics Core, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Weitzel JN; Division of Clinical Cancer Genomics, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Yuan YC; Division of Clinical Cancer Genomics, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Awasthi M; Bioinformatics Core, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Srivastava SK; Diabetes and Metabolism Research Institute, City of Hope Comprehensive Cancer Center, Duarte, CA 91010.
  • Awasthi YC; Department of Biochemistry and Molecular Biology, The University of Texas Medical Branch, Galveston, TX 77555-0647.
  • Singhal SS; Department of Biochemistry and Molecular Biology, The University of Texas Medical Branch, Galveston, TX 77555-0647.
Proc Natl Acad Sci U S A ; 115(15): 3918-3923, 2018 04 10.
Article en En | MEDLINE | ID: mdl-29572430
TP53 (p53) is a tumor suppressor whose functions are lost or altered in most malignancies. p53 homozygous knockout (p53-/-) mice uniformly die of spontaneous malignancy, typically T-cell lymphoma. RALBP1 (RLIP76, Rlip) is a stress-protective, mercapturic acid pathway transporter protein that also functions as a Ral effector involved in clathrin-dependent endocytosis. In stark contrast to p53-/- mice, Rlip-/- mice are highly resistant to carcinogenesis. We report here that partial Rlip deficiency induced by weekly administration of an Rlip-specific phosphorothioate antisense oligonucleotide, R508, strongly inhibited spontaneous as well as benzo(a)pyrene-induced carcinogenesis in p53-/- mice. This treatment effectively prevented large-scale methylomic and transcriptomic abnormalities suggestive of inflammation found in cancer-bearing p53-/- mice. The remarkable efficiency with which Rlip deficiency suppresses spontaneous malignancy in p53-/- mice has not been observed with any previously reported pharmacologic or genetic intervention. These findings are supported by cross-breeding experiments demonstrating that hemizygous Rlip deficiency also reduces the spontaneous malignancy phenotype of p53+/- mice. Rlip is found on the cell surface, and antibodies directed against Rlip were found to inhibit growth and promote apoptosis of cell lines as effectively as Rlip siRNA. The work presented here investigates several features, including oxidative DNA damage of the Rlip-p53 association in malignant transformation, and offers a paradigm for the mechanisms of tumor suppression by p53 and the prospects of suppressing spontaneous malignancy in hereditary cancer syndromes such as Li-Fraumeni.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteína p53 Supresora de Tumor / Proteínas Activadoras de GTPasa / Neoplasias Límite: Animals / Female / Humans / Male Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2018 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteína p53 Supresora de Tumor / Proteínas Activadoras de GTPasa / Neoplasias Límite: Animals / Female / Humans / Male Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2018 Tipo del documento: Article Pais de publicación: Estados Unidos