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Telomere attrition in heart failure: a flow-FISH longitudinal analysis of circulating monocytes.
Teubel, Iris; Elchinova, Elena; Roura, Santiago; Fernández, Marco A; Gálvez-Montón, Carolina; Moliner, Pedro; de Antonio, Marta; Lupón, Josep; Bayés-Genís, Antoni.
Afiliación
  • Teubel I; Flow Cytometry Facility, Germans Trias i Pujol Health Science Research Institute, Badalona, Spain.
  • Elchinova E; Cardiology Service, Germans Trias i Pujol University Hospital, Carretera del Canyet s/n, 08916, Badalona, Spain.
  • Roura S; Department of Medicine, Universitat Autònoma de Barcelona, Barcelona, Spain.
  • Fernández MA; ICREC Research Program, Germans Trias i Pujol Health Science Research Institute, Badalona, Spain.
  • Gálvez-Montón C; Center of Regenerative Medicinein Barcelona, Barcelona, Spain.
  • Moliner P; CIBERCV, Instituto de Salud Carlos III, Madrid, Spain.
  • de Antonio M; Flow Cytometry Facility, Germans Trias i Pujol Health Science Research Institute, Badalona, Spain.
  • Lupón J; ICREC Research Program, Germans Trias i Pujol Health Science Research Institute, Badalona, Spain.
  • Bayés-Genís A; CIBERCV, Instituto de Salud Carlos III, Madrid, Spain.
J Transl Med ; 16(1): 35, 2018 02 20.
Article en En | MEDLINE | ID: mdl-29463269
BACKGROUND: Cross-sectional investigations report shorter telomeres in patients with heart failure (HF); however, no studies describe telomere length (TL) trajectory and its relationship with HF progression. Here we aimed to investigate telomere shortening over time and its relationship to outcomes. METHODS: Our study cohort included 101 ambulatory patients with HF. Blood samples were collected at baseline (n = 101) and at the 1-year follow-up (n = 54). Using flow-FISH analysis of circulating monocytes, we simultaneously measured three monocyte subsets-classical (CD14++CD16-), intermediate (CD14++CD16+), and nonclassical (CD14+CD16++)-and their respective TLs based on FITC-labeled PNA probe hybridization. The primary endpoints were all-cause death and the composite of all-cause death or HF-related hospitalization, assessed at 2.3 ± 0.6 years. All statistical analyses were executed by using the SPSS 15.0 software, and included Student's t test and ANOVA with post hoc Scheffe analysis, Pearson or Spearman rho correlation and univariate Cox regression when applicable. RESULTS: We found high correlations between TL values of different monocyte subsets: CD14++CD16+ vs. CD14++CD16-, R = 0.95, p < 0.001; CD14++CD16+ vs. CD14+CD16++, R = 0.90, p < 0.001; and CD14++CD16- vs. CD14+CD16++, R = 0.89, p < 0.001. Mean monocyte TL exhibited significant attrition from baseline to the 1-year follow-up (11.1 ± 3.3 vs. 8.3 ± 2.1, p < 0.001). TL did not significantly differ between monocyte subsets at either sampling time-point (all p values > 0.1). Cox regression analyses did not indicate that TL or ΔTL was associated with all-cause death or the composite endpoint. CONCLUSIONS: Overall, this longitudinal study demonstrated a ~ 22% reduction of TL in monocytes from ambulatory patients with HF within 1 year. TL and ΔTL were not related to outcomes over long-term follow-up.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Monocitos / Telómero / Hibridación Fluorescente in Situ / Insuficiencia Cardíaca Tipo de estudio: Observational_studies / Risk_factors_studies Límite: Aged / Female / Humans / Male Idioma: En Revista: J Transl Med Año: 2018 Tipo del documento: Article País de afiliación: España Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Monocitos / Telómero / Hibridación Fluorescente in Situ / Insuficiencia Cardíaca Tipo de estudio: Observational_studies / Risk_factors_studies Límite: Aged / Female / Humans / Male Idioma: En Revista: J Transl Med Año: 2018 Tipo del documento: Article País de afiliación: España Pais de publicación: Reino Unido