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Bi-allelic mutations of CCDC88C are a rare cause of severe congenital hydrocephalus.
Ruggeri, Gaia; Timms, Andrew E; Cheng, Chi; Weiss, Avery; Kollros, Peter; Chapman, Teresa; Tully, Hannah; Mirzaa, Ghayda M.
Afiliación
  • Ruggeri G; Center for Integrative Brain Research, Seattle Children's Research Institute, Seattle, Washington.
  • Timms AE; Center for Developmental Biology and Regenerative Medicine, Seattle Children's Research Institute, Seattle, Washington.
  • Cheng C; Center for Integrative Brain Research, Seattle Children's Research Institute, Seattle, Washington.
  • Weiss A; Division of Ophthalmology, Roger H. Johnson Vision Lab, Seattle Children's Hospital, Seattle, Washington.
  • Kollros P; Department of Ophthalmology, University of Washington School of Medicine, Seattle, Washington.
  • Chapman T; Division of Pediatric Neurology, Seattle Children's Hospital, Seattle, Washington.
  • Tully H; Department of Radiology, Seattle Children's Hospital, Seattle, Washington.
  • Mirzaa GM; University of Washington School of Medicine, University of Washington, Seattle, Washington.
Am J Med Genet A ; 176(3): 676-681, 2018 03.
Article en En | MEDLINE | ID: mdl-29341397
Congenital or infantile hydrocephalus is caused by genetic and non-genetic factors and is highly heterogeneous in etiology. In recent studies, a limited number of genetic causes of hydrocephalus have been identified. To date, recessive mutations in the CCDC88C gene have been identified as a cause of non-syndromic congenital hydrocephalus in three reported families. Here, we report the fourth known family with two affected individuals with congenital hydrocephalus due to a homozygous mutation in the CCDC88C gene identified by whole exome sequencing. Our two newly described children, as well as the previously published ones, all shared several features including severe infantile-onset hydrocephalus, mild to severe intellectual delay, varying degrees of motor delay, and infantile onset seizures. All identified homozygous mutations in CCDC88C abolish the PDZ binding site necessary for proper CCDC88C protein function in the Wnt signaling pathway. Our report further establishes CCDC88C as one of the few known recessive causes of severe prenatal-onset hydrocephalus. Recognition of this syndrome has important diagnostic and genetic implications for families identified in the future.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fenotipo / Péptidos y Proteínas de Señalización Intracelular / Alelos / Hidrocefalia / Proteínas de Microfilamentos / Mutación Tipo de estudio: Prognostic_studies Límite: Female / Humans / Infant / Male Idioma: En Revista: Am J Med Genet A Asunto de la revista: GENETICA MEDICA Año: 2018 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fenotipo / Péptidos y Proteínas de Señalización Intracelular / Alelos / Hidrocefalia / Proteínas de Microfilamentos / Mutación Tipo de estudio: Prognostic_studies Límite: Female / Humans / Infant / Male Idioma: En Revista: Am J Med Genet A Asunto de la revista: GENETICA MEDICA Año: 2018 Tipo del documento: Article Pais de publicación: Estados Unidos