Your browser doesn't support javascript.
loading
Synergistic effect of l-ascorbic acid and hyaluronic acid on the expressions of matrix metalloproteinase-3 and -9 in human chondrocytes.
Huang, Teng-Le; Yang, Che-Hua; Yanai, Goichi; Liao, Jo-Yu; Sumi, Shoichiro; Yang, Kai-Chiang.
Afiliación
  • Huang TL; Department of Orthopedics, Tai-An Hospital Shuang Shi Branch, Taichung, 40455, Taiwan.
  • Yang CH; Department of Biomedical Imaging and Radiological Science, College of Medicine, China Medical University, Taichung, 40402, Taiwan.
  • Yanai G; Graduate Institute of Manufacturing Technology, National Taipei University of Technology, Taipei, 10608, Taiwan.
  • Liao JY; Laboratory of Organ and Tissue Reconstruction, Institute for Frontier Life and Medical Sciences, Kyoto University, 606-8507, Japan.
  • Sumi S; Department of Orthopedics, Tai-An Hospital Shuang Shi Branch, Taichung, 40455, Taiwan.
  • Yang KC; Department of Biomedical Imaging and Radiological Science, College of Medicine, China Medical University, Taichung, 40402, Taiwan.
J Biomed Mater Res B Appl Biomater ; 106(5): 1809-1817, 2018 07.
Article en En | MEDLINE | ID: mdl-28914997
Proinflammatory cytokines and reactive oxygen species (ROS) are known to be involved in the initiation and progression of osteoarthritis (OA). New evidence clarifying the correlation between ROS and inflammation has indicated that oxidative stress can up-regulate inflammatory cytokines. l-Ascorbic acid (AA), an antioxidant, has been shown to have anti-inflammatory effects and improve matrix deposition in chondrocytes. The purpose of this study was to examine the effects of hyaluronic acid (HA; 100 µg/mL) supplemented with AA (50 µg/mL) on human normal and interleukin-1 beta-stimulated (IL-1ß, 10 ng/mL) chondrocytes. HA, AA, and HA + AA treatment did not change cell morphology, viability, proliferation, and glycosaminoglycan production in normal chondrocytes. HA, AA, and HA + AA, by contrast, partially restored viability and morphology of hypertrophic chondrocytes, and HA and HA + AA further decreased the cytotoxicity of IL-1ß. Real-time PCR revealed that AA and HA + AA had no substantial effects on unstimulated chondrocytes, except for down-regulation of matrix metalloproteinase (MMP)-9 mRNA levels. For IL-1ß-stimulated chondrocytes, significant down-regulation of IL-1ß, tumor necrosis factor-alpha (TNF-α), MMP-3, and MMP-9 mRNA expression was found when cells were cultured in HA-supplemented media. Moreover, HA + AA supplementation further significantly decreased MMP-3 and MMP-9 mRNA expression. The protein production of MMP-3 was decreased, with a significant difference between the HA + AA group and HA group. The antioxidant capacity and superoxide dismutases activity were also partially restored in stimulated chondrocytes. HA supplemented with AA modulates MMPs expression and antioxidant fuction in chondrocytes. AA may enhance the anticatabolic effects of HA on OA chondrocytes. © 2017 Wiley Periodicals, Inc. J Biomed Mater Res Part B: Appl Biomater, 106B: 1809-1817, 2018.
Asunto(s)
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Osteoartritis / Ácido Ascórbico / Regulación Enzimológica de la Expresión Génica / Metaloproteinasa 3 de la Matriz / Condrocitos / Metaloproteinasa 9 de la Matriz / Ácido Hialurónico Límite: Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: J Biomed Mater Res B Appl Biomater Asunto de la revista: ENGENHARIA BIOMEDICA Año: 2018 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Osteoartritis / Ácido Ascórbico / Regulación Enzimológica de la Expresión Génica / Metaloproteinasa 3 de la Matriz / Condrocitos / Metaloproteinasa 9 de la Matriz / Ácido Hialurónico Límite: Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: J Biomed Mater Res B Appl Biomater Asunto de la revista: ENGENHARIA BIOMEDICA Año: 2018 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Estados Unidos