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Neuroprotection in Glaucoma: Animal Models and Clinical Trials.
Almasieh, Mohammadali; Levin, Leonard A.
Afiliación
  • Almasieh M; Departments of Ophthalmology and Neurology, McGill University, Montreal H4A 3S5, Canada; email: leonard.levin@mcgill.ca.
  • Levin LA; Maisonneuve-Rosemont Hospital Research Center and Department of Ophthalmology, University of Montreal, Montreal H1T 2M4, Canada.
Annu Rev Vis Sci ; 3: 91-120, 2017 09 15.
Article en En | MEDLINE | ID: mdl-28731838
Glaucoma is a progressive neurodegenerative disease that frequently results in irreversible blindness. Glaucoma causes death of retinal ganglion cells (RGCs) and their axons in the optic nerve, resulting in visual field deficits and eventual loss of visual acuity. Glaucoma is a complex optic neuropathy, and a successful strategy for its treatment requires not only better management of known risk factors such as elevated intraocular pressure and the development of improved tools for detecting RGC injury but also treatments that address this injury (i.e., neuroprotection). Experimental models of glaucoma provide insight into the cellular and molecular mechanisms of glaucomatous optic neuropathy and aid the development of neuroprotective therapies.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedades del Nervio Óptico / Glaucoma / Fármacos Neuroprotectores / Enfermedades Neurodegenerativas Tipo de estudio: Etiology_studies / Prognostic_studies / Risk_factors_studies Límite: Animals / Humans Idioma: En Revista: Annu Rev Vis Sci Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedades del Nervio Óptico / Glaucoma / Fármacos Neuroprotectores / Enfermedades Neurodegenerativas Tipo de estudio: Etiology_studies / Prognostic_studies / Risk_factors_studies Límite: Animals / Humans Idioma: En Revista: Annu Rev Vis Sci Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos