Your browser doesn't support javascript.
loading
PAX6 allelic heterogeneity in Mexican congenital aniridia patients: expanding the mutational spectrum with seven novel pathogenic variants.
Pérez-Solórzano, Sofía; Chacón-Camacho, Oscar F; Astiazarán, Mirena C; Ledesma-Gil, Gerardo; Zenteno, Juan Carlos.
Afiliación
  • Pérez-Solórzano S; Genetics Department, Institute of Ophthalmology 'Conde de Valenciana', Mexico City, Mexico.
  • Chacón-Camacho OF; Genetics Department, Institute of Ophthalmology 'Conde de Valenciana', Mexico City, Mexico.
  • Astiazarán MC; Genetics Department, Institute of Ophthalmology 'Conde de Valenciana', Mexico City, Mexico.
  • Ledesma-Gil G; Retina Department, Institute of Ophthalmology 'Conde de Valenciana', Mexico City, Mexico.
  • Zenteno JC; Genetics Department, Institute of Ophthalmology 'Conde de Valenciana', Mexico City, Mexico.
Clin Exp Ophthalmol ; 45(9): 875-883, 2017 12.
Article en En | MEDLINE | ID: mdl-28488383
IMPORTANCE: The importance of the study was to describe the clinical characteristics and mutational analysis of Mexican patients with aniridia. BACKGROUND: Aniridia is a panocular hereditary eye disease caused by mutations in the PAX6 transcription factor. Mutation detection rate is highly variable ranging from 30% to 90% in different populations. Very few studies have been published about the PAX6 mutational analysis in aniridia patients from Mexico. In order to establish a more representative PAX6 mutational frequency in the country, a cohort of 22 Mexican unrelated aniridia probands were analysed in this study. DESIGN: Case series. PARTICIPANTS: A total of 22 Mexican probands with bilateral isolated aniridia and their available relatives were included. METHODS: Sanger sequencing was used for the mutational analysis of all coding exons and flanking intronic regions of PAX6. MAIN OUTCOME MEASURES: Clinical characteristics and results of PAX6 mutational analysis in probands with aniridia and available family members. RESULTS: Molecular analysis of PAX6 in 22 index cases with aniridia allowed the identification of a total of 16 different mutations. Seven of these pathogenic variants are novel, including c.183C>G, p.(Y61*); c.718delC, p.(R240Efs*3); c.1149_1152delTCAG, p.(P385Wfs*139); c.257_266delAAATAGCCCA, p.(K86Sfs*35); c.836_843dupGCAACACA p.(P282Afs*86); c.1032+2_1032+3insT; and c.141+2T>A. Inter and intrafamilial phenotypic heterogeneity was found. CONCLUSIONS AND RELEVANCE: The mutational diagnostic rate in this series was 77%, which is comparable with reports from other populations. Importantly, no founder mutations were identified in this case series. Our results add 7 novel PAX6 pathogenic variants to the aniridia-related mutational spectrum and reveal considerable PAX6 allelic heterogeneity in this population.
Asunto(s)
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: ADN / Aniridia / Factor de Transcripción PAX6 / Mutación Tipo de estudio: Incidence_studies / Prognostic_studies Límite: Adolescent / Adult / Child / Child, preschool / Female / Humans / Male / Middle aged País/Región como asunto: Mexico Idioma: En Revista: Clin Exp Ophthalmol Asunto de la revista: OFTALMOLOGIA Año: 2017 Tipo del documento: Article País de afiliación: México Pais de publicación: Australia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: ADN / Aniridia / Factor de Transcripción PAX6 / Mutación Tipo de estudio: Incidence_studies / Prognostic_studies Límite: Adolescent / Adult / Child / Child, preschool / Female / Humans / Male / Middle aged País/Región como asunto: Mexico Idioma: En Revista: Clin Exp Ophthalmol Asunto de la revista: OFTALMOLOGIA Año: 2017 Tipo del documento: Article País de afiliación: México Pais de publicación: Australia