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Protective effects of Garcinol against neuropathic pain - Evidence from in vivo and in vitro studies.
Wang, Yi-Wei; Zhang, Xiang; Chen, Chun-Long; Liu, Qing-Zhen; Xu, Jia-Wen; Qian, Qing-Qing; Li, Wei-Yan; Qian, Yan-Ning.
Afiliación
  • Wang YW; Department of Anesthesiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing 210002, PR China.
  • Zhang X; Department of Anesthesiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing 210002, PR China.
  • Chen CL; Department of Anesthesiology, Nanjing General Hospital, Nanjing 210002, PR China.
  • Liu QZ; Department of Anesthesiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing 210002, PR China; Department of Anesthesiology, Nanjing General Hospital, Nanjing 210002, PR China.
  • Xu JW; Department of Anesthesiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing 210002, PR China.
  • Qian QQ; Department of Anesthesiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing 210002, PR China.
  • Li WY; Department of Anesthesiology, Nanjing General Hospital, Nanjing 210002, PR China. Electronic address: weiyanlee65@163.com.
  • Qian YN; Department of Anesthesiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing 210002, PR China. Electronic address: yanning_qian@163.com.
Neurosci Lett ; 647: 85-90, 2017 04 24.
Article en En | MEDLINE | ID: mdl-28302538
Neuroinflammatory processes have a vital role in the pathogenesis of neuropathic pain. Garcinol, harvested from Garcinia indica, is known to exert potent anti-inflammatory properties. Recent studies have indicated that Garcinol may inhibit activation of nuclear factor-κB (NF-κB) by inhibiting NF-κB/p65 acetylation. These findings prompted us to evaluate the protective effects of Garcinol in the lumbar fifth spinal nerve ligation (SNL)-induced rat model of neuropathic pain and Lipopolysaccharide(LPS)-stimulated primary cultured microglia. In the present study, we found that intrathecal administration of Garcinol significantly attenuated SNL-induced nociceptive behaviors. Garcinol suppressed microglial activation as well as the expression of interleukin (IL)-1ß, IL-6, inducible nitric oxide synthase (iNOS)/nitric oxide (NO), and cyclooxygenase-2 (COX-2)/prostaglandin E2 (PGE2) in the spinal cord of SNL rats. It also reduced the nuclear translocation of NF-κB by decreasing acetyl-p65 protein expression. Similarly, in the in vitro study, Garcinol decreased the production of NO/iNOS, PGE2/COX-2, and proinflammatory cytokines in LPS-exposed microglia. Likewise, Garcinol inhibited the NF-κB signaling pathway by downregulating acetyl-p65 levels in LPS-challenged microglia. Our findings suggest that Garcinol may have protective effects against neuropathic pain that are associated with the inhibition of neuroinflammation in microglia. Therefore, Garcinol could be a promising agent in the treatment of neuropathic pain.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Terpenos / Antiinflamatorios no Esteroideos / Microglía / Neuralgia Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Neurosci Lett Año: 2017 Tipo del documento: Article Pais de publicación: Irlanda

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Terpenos / Antiinflamatorios no Esteroideos / Microglía / Neuralgia Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Neurosci Lett Año: 2017 Tipo del documento: Article Pais de publicación: Irlanda