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FZR1 loss increases sensitivity to DNA damage and consequently promotes murine and human B-cell acute leukemia.
Ishizawa, Jo; Sugihara, Eiji; Kuninaka, Shinji; Mogushi, Kaoru; Kojima, Kensuke; Benton, Christopher B; Zhao, Ran; Chachad, Dhruv; Hashimoto, Norisato; Jacamo, Rodrigo O; Qiu, Yihua; Yoo, Suk Young; Okamoto, Shinichiro; Andreeff, Michael; Kornblau, Steven M; Saya, Hideyuki.
Afiliación
  • Ishizawa J; Division of Gene Regulation, Institute for Advanced Medical Research, and.
  • Sugihara E; Division of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
  • Kuninaka S; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Mogushi K; Division of Gene Regulation, Institute for Advanced Medical Research, and.
  • Kojima K; Division of Gene Regulation, Institute for Advanced Medical Research, and.
  • Benton CB; Intractable Disease Research Center, Graduate School of Medicine, Juntendo University, Tokyo, Japan.
  • Zhao R; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Chachad D; Hematology, Respiratory Medicine, and Oncology, Department of Medicine, Saga University, Saga, Japan; and.
  • Hashimoto N; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Jacamo RO; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Qiu Y; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Yoo SY; Division of Gene Regulation, Institute for Advanced Medical Research, and.
  • Okamoto S; Division of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
  • Andreeff M; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Kornblau SM; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX.
  • Saya H; Department of Bioinformatics and Computational Biology, The University of Texas MD Anderson Cancer Center, Houston, TX.
Blood ; 129(14): 1958-1968, 2017 04 06.
Article en En | MEDLINE | ID: mdl-28143883
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Daño del ADN / Leucemia-Linfoma Linfoblástico de Células Precursoras B / Regulación Leucémica de la Expresión Génica / Proteínas Cdh1 / Proteínas de Neoplasias Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Blood Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Daño del ADN / Leucemia-Linfoma Linfoblástico de Células Precursoras B / Regulación Leucémica de la Expresión Génica / Proteínas Cdh1 / Proteínas de Neoplasias Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Blood Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos