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Macrophage Transcriptional Profile Identifies Lipid Catabolic Pathways That Can Be Therapeutically Targeted after Spinal Cord Injury.
Zhu, Y; Lyapichev, K; Lee, D H; Motti, D; Ferraro, N M; Zhang, Y; Yahn, S; Soderblom, C; Zha, J; Bethea, J R; Spiller, K L; Lemmon, V P; Lee, J K.
Afiliación
  • Zhu Y; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Lyapichev K; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Lee DH; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Motti D; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Ferraro NM; School of Biomedical Engineering, Science and Health Systems, Drexel University, Philadelphia, Pennsylvania 19104.
  • Zhang Y; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Yahn S; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Soderblom C; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Zha J; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Bethea JR; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Spiller KL; School of Biomedical Engineering, Science and Health Systems, Drexel University, Philadelphia, Pennsylvania 19104.
  • Lemmon VP; Miami Project to Cure Paralysis, Department of Neurological Surgery, School of Medicine and.
  • Lee JK; Center for Computational Science, University of Miami, Miami, Florida 33136 and.
J Neurosci ; 37(9): 2362-2376, 2017 03 01.
Article en En | MEDLINE | ID: mdl-28130359
Although infiltrating macrophages influence many pathological processes after spinal cord injury (SCI), the intrinsic molecular mechanisms that regulate their function are poorly understood. A major hurdle has been dissecting macrophage-specific functions from those in other cell types as well as understanding how their functions change over time. Therefore, we used the RiboTag method to obtain macrophage-specific mRNA directly from the injured spinal cord in mice and performed RNA sequencing to investigate their transcriptional profile. Our data show that at 7 d after SCI, macrophages are best described as foam cells, with lipid catabolism representing the main biological process, and canonical nuclear receptor pathways as their potential mediators. Genetic deletion of a lipoprotein receptor, CD36, reduces macrophage lipid content and improves lesion size and locomotor recovery. Therefore, we report the first macrophage-specific transcriptional profile after SCI and highlight the lipid catabolic pathway as an important macrophage function that can be therapeutically targeted after SCI.SIGNIFICANCE STATEMENT The intrinsic molecular mechanisms that regulate macrophage function after spinal cord injury (SCI) are poorly understood. We obtained macrophage-specific mRNA directly from the injured spinal cord and performed RNA sequencing to investigate their transcriptional profile. Our data show that at 7 d after SCI, macrophages are best described as foam cells, with lipid catabolism representing the main biological process and canonical nuclear receptor pathways as their potential mediators. Genetic deletion of a lipoprotein receptor, CD36, reduces macrophage lipid content and improves lesion size and locomotor recovery. Therefore, we report the first macrophage-specific transcriptional profile after SCI and highlight the lipid catabolic pathway as an important macrophage function that can be therapeutically targeted after SCI.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Traumatismos de la Médula Espinal / Metabolismo de los Lípidos / Macrófagos Límite: Animals Idioma: En Revista: J Neurosci Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Traumatismos de la Médula Espinal / Metabolismo de los Lípidos / Macrófagos Límite: Animals Idioma: En Revista: J Neurosci Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos