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Eosinophils are required to suppress Th2 responses in Peyer's patches during intestinal infection by nematodes.
Strandmark, J; Steinfelder, S; Berek, C; Kühl, A A; Rausch, S; Hartmann, S.
Afiliación
  • Strandmark J; Institute of Immunology, Department of Veterinary Medicine, Freie Universität Berlin, Berlin, Germany.
  • Steinfelder S; Institute of Immunology, Department of Veterinary Medicine, Freie Universität Berlin, Berlin, Germany.
  • Berek C; Deutsches Rheuma Forschungszentrum Berlin, a Leibniz Institute, Berlin, Germany.
  • Kühl AA; Department of Internal Medicine, Rheumatology and Clinical Immunology/Research Center ImmunoSciences (RCIS), Charité-University Medicine Berlin, Berlin, Germany.
  • Rausch S; Institute of Immunology, Department of Veterinary Medicine, Freie Universität Berlin, Berlin, Germany.
  • Hartmann S; Institute of Immunology, Department of Veterinary Medicine, Freie Universität Berlin, Berlin, Germany.
Mucosal Immunol ; 10(3): 661-672, 2017 05.
Article en En | MEDLINE | ID: mdl-27805618
Infections with enteric nematodes result in systemic type 2 helper T (Th2) responses, expansion of immunoglobulin (Ig)G1 antibodies, and eosinophilia. Eosinophils have a supportive role in mucosal Th2 induction during airway hyperreactivity. Whether eosinophils affect the local T-cell and antibody response in the gut-associated lymphoid tissue during enteric infections is unknown. We infected eosinophil-deficient ΔdblGATA-1 mice with the Th2-inducing small intestinal nematode Heligmosomoides polygyrus and found that parasite fecundity was decreased in the absence of eosinophils. A lack of eosinophils resulted in significantly augmented expression of GATA-3 and IL-4 by CD4+ T cells during acute infection, a finding strictly limited to Peyer's patches (PP). The increase in IL-4-producing cells in ΔdblGATA-1 mice was particularly evident within the CXCR5+PD-1+ T-follicular helper cell population and was associated with a switch of germinal centre B cells to IgG1 production and elevated serum IgG1 levels. In contrast, infected wild-type mice had a modest IgG1 response in the PP, whereas successfully maintaining a population of IgA+ germinal center B cells. Our results suggest a novel role for eosinophils during intestinal infection whereby they restrict IL-4 responses by follicular T helper cells and IgG1 class switching in the PP to ensure maintenance of local IgA production.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ganglios Linfáticos Agregados / Nematospiroides dubius / Linfocitos B / Infecciones por Strongylida / Células Th2 / Eosinófilos / Intestinos Límite: Animals Idioma: En Revista: Mucosal Immunol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2017 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ganglios Linfáticos Agregados / Nematospiroides dubius / Linfocitos B / Infecciones por Strongylida / Células Th2 / Eosinófilos / Intestinos Límite: Animals Idioma: En Revista: Mucosal Immunol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2017 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos