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The Association Between Retinal Neuronal Layer and Brain Structure is Disrupted in Patients with Cognitive Impairment and Alzheimer's Disease.
Liu, Siwei; Ong, Yi-Ting; Hilal, Saima; Loke, Yng Miin; Wong, Tien Y; Chen, Christopher Li-Hsian; Cheung, Carol Y; Zhou, Juan.
Afiliación
  • Liu S; Center for Cognitive Neuroscience, Neuroscience and Behavioral Disorders Program, Duke-National University of Singapore Medical School, Singapore.
  • Ong YT; Singapore Eye Research Institute, Singapore National Eye Centre, Singapore.
  • Hilal S; Department of Pharmacology, National University of Singapore, Singapore.
  • Loke YM; Memory Aging & Cognition Centre, National University Health System, Singapore.
  • Wong TY; Center for Cognitive Neuroscience, Neuroscience and Behavioral Disorders Program, Duke-National University of Singapore Medical School, Singapore.
  • Chen CL; Singapore Eye Research Institute, Singapore National Eye Centre, Singapore.
  • Cheung CY; Department of Pharmacology, National University of Singapore, Singapore.
  • Zhou J; Memory Aging & Cognition Centre, National University Health System, Singapore.
J Alzheimers Dis ; 54(2): 585-95, 2016 09 06.
Article en En | MEDLINE | ID: mdl-27567815
Both healthy and pathological aging due to Alzheimer's disease (AD) are associated with decreased brain grey matter volume (GMV) and disrupted white matter (WM) microstructure. Thinner macular ganglion cell-inner plexiform layer (GC-IPL) measured by spectral-domain optical coherence tomography has been reported in patients with AD and mild cognitive impairment. Emerging evidence suggested a link between thinner GC-IPL and lower GMV in subjects with no dementia using region-of-interest-based approach. However, it remains unknown whether GC-IPL thickness is associated with brain WM microstructure and how such association differed between normal and cognitively impaired subjects. Here, for subjects with no cognitive impairment (NCI), thinner GC-IPL was associated with lower WM microstructure integrity in the superior longitudinal fasciculus, inferior fronto-occipital fasciculus, corticospinal tracts, anterior thalamic radiation, and cingulum regions, while it was weakly associated with lower GMV in visual cortex and cerebellum. Nevertheless, these retina-brain associations were disrupted in the presence of cognitive impairment. Correlations between GMV and GC-IPL were lost in patients with cognitive impairment but no dementia (CIND) and AD patients. GC-IPL was related to WM microstructural disruption in similar regions with decreased significance. In contrast, lower WM microstructure integrity in the fornix showed a trend of correlation with thinner GC-IPL in both CIND and AD but not NCI. Collectively, our findings suggest the possible physiological retina-brain relationship in healthy aging, which might be disrupted by disease-induced changes in patients with cognitive impairment. Longitudinal study with larger patient sample should follow to confirm the disease mechanism behind these retina-brain relationship changes.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Encéfalo / Neuronas Retinianas / Enfermedad de Alzheimer / Disfunción Cognitiva Tipo de estudio: Observational_studies / Risk_factors_studies Límite: Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: J Alzheimers Dis Asunto de la revista: GERIATRIA / NEUROLOGIA Año: 2016 Tipo del documento: Article País de afiliación: Singapur Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Encéfalo / Neuronas Retinianas / Enfermedad de Alzheimer / Disfunción Cognitiva Tipo de estudio: Observational_studies / Risk_factors_studies Límite: Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: J Alzheimers Dis Asunto de la revista: GERIATRIA / NEUROLOGIA Año: 2016 Tipo del documento: Article País de afiliación: Singapur Pais de publicación: Países Bajos