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The majority of murine γδ T cells at the maternal-fetal interface in pregnancy produce IL-17.
Pinget, Gabriela V; Corpuz, Theresa M; Stolp, Jessica; Lousberg, Erin L; Diener, Kerrilyn R; Robertson, Sarah A; Sprent, Jonathan; Webster, Kylie E.
Afiliación
  • Pinget GV; Immunology Division, Garvan Institute of Medical Research, Darlinghurst, NSW, Australia.
  • Corpuz TM; Immunology Division, Garvan Institute of Medical Research, Darlinghurst, NSW, Australia.
  • Stolp J; Immunology Division, Garvan Institute of Medical Research, Darlinghurst, NSW, Australia.
  • Lousberg EL; The Robinson Research Institute and School of Medicine, University of Adelaide, Adelaide, SA, Australia.
  • Diener KR; Experimental Therapeutics Laboratory, Hanson Institute, Royal Adelaide Hospital, and Sansom Institute, School of Pharmacy and Medical Science, University of South Australia, Adelaide, SA, Australia.
  • Robertson SA; The Robinson Research Institute and School of Medicine, University of Adelaide, Adelaide, SA, Australia.
  • Sprent J; Experimental Therapeutics Laboratory, Hanson Institute, Royal Adelaide Hospital, and Sansom Institute, School of Pharmacy and Medical Science, University of South Australia, Adelaide, SA, Australia.
  • Webster KE; The Robinson Research Institute and School of Medicine, University of Adelaide, Adelaide, SA, Australia.
Immunol Cell Biol ; 94(7): 623-30, 2016 08.
Article en En | MEDLINE | ID: mdl-27241697
Compared with lymphoid tissues, the immune cell compartment at mucosal sites is enriched with T cells bearing the γδ T-cell receptor (TCR). The female reproductive tract, along with the placenta and uterine decidua during pregnancy, are populated by γδ T cells predominantly expressing the invariant Vγ6(+)Vδ1(+) receptor. Surprisingly little is understood about the function of these cells. We found that the majority of γδ T cells in the non-pregnant uterus, pregnant uterus, decidua and placenta of mice express the transcription factor RORγt and produce interleukin-17 (IL-17). In contrast, IFNγ-producing γδ T cells were markedly reduced in gestational tissues compared with uterine-draining lymph nodes and spleen. Both uterine-resident invariant Vγ6(+) and Vγ4(+) γδ T cells which are more typically found in lymphoid tissues and circulating blood, were found to express IL-17. Vγ4(+) γδ T cells were particularly enriched in the placenta, suggesting a pregnancy-specific recruitment or expansion of these cells. A small increase in IL-17-producing γδ T cells was observed in allogeneic compared with syngeneic pregnancy, suggesting a contribution to regulating the maternal response to paternally-derived alloantigens. However, their high proportions also in non-pregnant uteri and gestational tissues of syngeneic pregnancy imply a role in the prevention of intrauterine infection or quality control of fetal development. These data suggest the need for a more rigorous evaluation of the role of IL-17 in sustaining normal pregnancy, particularly as emerging data points to a pathogenic role for IL-17 in pre-eclampsia, pre-term birth, miscarriage and maternal immune activation-induced behavioral abnormalities in offspring.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos T / Receptores de Antígenos de Linfocitos T gamma-delta / Interleucina-17 / Intercambio Materno-Fetal Límite: Animals / Pregnancy Idioma: En Revista: Immunol Cell Biol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2016 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos T / Receptores de Antígenos de Linfocitos T gamma-delta / Interleucina-17 / Intercambio Materno-Fetal Límite: Animals / Pregnancy Idioma: En Revista: Immunol Cell Biol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2016 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Estados Unidos