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Complement inhibition by hydroxychloroquine prevents placental and fetal brain abnormalities in antiphospholipid syndrome.
Bertolaccini, Maria Laura; Contento, Gregorio; Lennen, Ross; Sanna, Giovanni; Blower, Philip J; Ma, Michelle T; Sunassee, Kavitha; Girardi, Guillermina.
Afiliación
  • Bertolaccini ML; Division of Women's Health, St Thomas' Hospital, King's College London, London, SE1 7EH, UK.
  • Contento G; Division of Women's Health, St Thomas' Hospital, King's College London, London, SE1 7EH, UK.
  • Lennen R; BHF/University Centre for Cardiovascular Science, University of Edinburgh, Edinburgh, EH16 4TJ, UK.
  • Sanna G; Louise Coote Lupus Unit, Guy's and St Thomas' NHS Foundation Trust, St Thomas' Hospital, London, SE1 7EH, UK.
  • Blower PJ; Division of Imaging Sciences and Biomedical Engineering, King's College London, London, SE1 7EH, UK.
  • Ma MT; Division of Imaging Sciences and Biomedical Engineering, King's College London, London, SE1 7EH, UK.
  • Sunassee K; Division of Imaging Sciences and Biomedical Engineering, King's College London, London, SE1 7EH, UK.
  • Girardi G; Division of Women's Health, St Thomas' Hospital, King's College London, London, SE1 7EH, UK; MRC Centre for Inflammation Research, Queen's Medical Research Institute University of Edinburgh, Edinburgh, EH16 4TJ, UK. Electronic address: guillermina.girardi@kcl.ac.uk.
J Autoimmun ; 75: 30-38, 2016 Dec.
Article en En | MEDLINE | ID: mdl-27160365
Placental ischemic disease and adverse pregnancy outcomes are frequently observed in patients with antiphospholipid syndrome (APS). Despite the administration of conventional antithrombotic treatment a significant number of women continue to experience adverse pregnancy outcomes, with uncertain prevention and management. Efforts to develop effective pharmacological strategies for refractory obstetric APS cases will be of significant clinical benefit for both mothers and fetuses. Although the antimalarial drug, hydroxychloroquine (HCQ) is increasingly used to treat pregnant women with APS, little is known about its efficacy and mechanism of action of HCQ. Because complement activation plays a crucial and causative role in placental ischemia and abnormal fetal brain development in APS we hypothesised that HCQ prevents these pregnancy complications through inhibition of complement activation. Using a mouse model of obstetric APS that closely resembles the clinical condition, we found that HCQ prevented fetal death and the placental metabolic changes -measured by proton magnetic resonance spectroscopy in APS-mice. Using 111In labelled antiphospholipid antibodies (aPL) we identified the placenta and the fetal brain as the main organ targets in APS-mice. Using this same method, we found that HCQ does not inhibit aPL binding to tissues as was previously suggested from in vitro studies. While HCQ did not affect aPL binding to fetal brain it prevented fetal brain abnormal cortical development. HCQ prevented complement activation in vivo and in vitro. Complement C5a levels in serum samples from APS patients and APS-mice were lower after treatment with HCQ while the antibodies titres remained unchanged. HCQ prevented not only placental insufficiency but also abnormal fetal brain development in APS. By inhibiting complement activation, HCQ might also be an effective antithrombotic therapy.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Placenta / Complicaciones del Embarazo / Encéfalo / Síndrome Antifosfolípido / Activación de Complemento / Hidroxicloroquina Límite: Adult / Animals / Female / Humans / Male / Middle aged / Pregnancy Idioma: En Revista: J Autoimmun Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2016 Tipo del documento: Article Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Placenta / Complicaciones del Embarazo / Encéfalo / Síndrome Antifosfolípido / Activación de Complemento / Hidroxicloroquina Límite: Adult / Animals / Female / Humans / Male / Middle aged / Pregnancy Idioma: En Revista: J Autoimmun Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2016 Tipo del documento: Article Pais de publicación: Reino Unido