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The polyphenols resveratrol and epigallocatechin-3-gallate restore the severe impairment of mitochondria in hippocampal progenitor cells from a Down syndrome mouse model.
Valenti, Daniela; de Bari, Lidia; de Rasmo, Domenico; Signorile, Anna; Henrion-Caude, Alexandra; Contestabile, Andrea; Vacca, Rosa Anna.
Afiliación
  • Valenti D; Institute of Biomembranes and Bioenergetics, National Council of Research, Bari, Italy. Electronic address: d.valenti@ibbe.cnr.it.
  • de Bari L; Institute of Biomembranes and Bioenergetics, National Council of Research, Bari, Italy.
  • de Rasmo D; Institute of Biomembranes and Bioenergetics, National Council of Research, Bari, Italy.
  • Signorile A; Department of Basic Medical Sciences, Neuroscience and Sense Organs, University of Bari, Italy.
  • Henrion-Caude A; INSERM UMR1163, Université Paris Descartes, Institut Imagine, Paris, France; Hôpital Necker-Enfants Malades, Paris, France.
  • Contestabile A; Neuroscience and Brain Technologies Department, Istituto Italiano di Tecnologia, Genova, Italy.
  • Vacca RA; Institute of Biomembranes and Bioenergetics, National Council of Research, Bari, Italy. Electronic address: r.vacca@ibbe.cnr.it.
Biochim Biophys Acta ; 1862(6): 1093-104, 2016 06.
Article en En | MEDLINE | ID: mdl-26964795
Mitochondrial dysfunctions critically impair nervous system development and are potentially involved in the pathogenesis of various neurodevelopmental disorders, including Down syndrome (DS), the most common genetic cause of intellectual disability. Previous studies from our group demonstrated impaired mitochondrial activity in peripheral cells from DS subjects and the efficacy of epigallocatechin-3-gallate (EGCG) - a natural polyphenol major component of green tea - to counteract the mitochondrial energy deficit. In this study, to gain insight into the possible role of mitochondria in DS intellectual disability, mitochondrial functions were analyzed in neural progenitor cells (NPCs) isolated from the hippocampus of Ts65Dn mice, a widely used model of DS which recapitulates many major brain structural and functional phenotypes of the syndrome, including impaired hippocampal neurogenesis. We found that, during NPC proliferation, mitochondrial bioenergetics and mitochondrial biogenic program were strongly compromised in Ts65Dn cells, but not associated with free radical accumulation. These data point to a central role of mitochondrial dysfunction as an inherent feature of DS and not as a consequence of cell oxidative stress. Further, we disclose that, besides EGCG, also the natural polyphenol resveratrol, which displays a neuroprotective action in various human diseases but never tested in DS, restores oxidative phosphorylation efficiency and mitochondrial biogenesis, and improves proliferation of NPCs. These effects were associated with the activation of PGC-1α/Sirt1/AMPK axis by both polyphenols. This research paves the way for using nutraceuticals as a potential therapeutic tool in preventing or managing some energy deficit-associated DS clinical manifestations.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Catequina / Síndrome de Down / Neurogénesis / Células-Madre Neurales / Resveratrol / Hipocampo / Mitocondrias / Antioxidantes Límite: Animals Idioma: En Revista: Biochim Biophys Acta Año: 2016 Tipo del documento: Article Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Catequina / Síndrome de Down / Neurogénesis / Células-Madre Neurales / Resveratrol / Hipocampo / Mitocondrias / Antioxidantes Límite: Animals Idioma: En Revista: Biochim Biophys Acta Año: 2016 Tipo del documento: Article Pais de publicación: Países Bajos