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Loss of diacylglycerol kinase epsilon in mice causes endothelial distress and impairs glomerular Cox-2 and PGE2 production.
Zhu, Jili; Chaki, Moumita; Lu, Dongmei; Ren, Chongyu; Wang, Shan-Shan; Rauhauser, Alysha; Li, Binghua; Zimmerman, Susan; Jun, Bokkyoo; Du, Yong; Vadnagara, Komal; Wang, Hanquin; Elhadi, Sarah; Quigg, Richard J; Topham, Matthew K; Mohan, Chandra; Ozaltin, Fatih; Zhou, Xin J; Marciano, Denise K; Bazan, Nicolas G; Attanasio, Massimo.
Afiliación
  • Zhu J; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas; Department of Nephrology, Renmin Hospital, Wuhan University, Hubei, Wuhan, China;
  • Chaki M; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas; Department of Neuroscience, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Lu D; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Ren C; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Wang SS; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Rauhauser A; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Li B; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Zimmerman S; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Jun B; Department of Neuroscience, Louisiana State University, New Orleans, Louisiana;
  • Du Y; Biomedical Engineering, University of Houston, Houston, Texas;
  • Vadnagara K; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Wang H; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas; Institute of Basic Medical Sciences, Hubei University of Medicine, Hubei, Shiyan, China;
  • Elhadi S; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas; Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Quigg RJ; Department of Medicine, University of Buffalo, Buffalo, New York;
  • Topham MK; Huntsman Cancer Institute, University of Utah, Salt Lake City, Utah;
  • Mohan C; Biomedical Engineering, University of Houston, Houston, Texas;
  • Ozaltin F; Department of Pediatric Nephrology, Faculty of Medicine, Hacettepe University, Ankara, Turkey; Nephrogenetics Laboratory, Department of Pediatric Nephrology, Faculty of Medicine, Hacettepe University, Ankara, Turkey;
  • Zhou XJ; Renal Path Diagnostics, Pathologist BioMedical Laboratories and Department of Pathology, Baylor University Medical Center, Dallas, Texas; and.
  • Marciano DK; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas;
  • Bazan NG; Department of Neuroscience, Louisiana State University, New Orleans, Louisiana;
  • Attanasio M; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas; Eugene McDermott Center for Growth and Development, The University of Texas Southwestern Medical Center, Dallas, Texas massimo.attanasio@utsouthwestern.edu.
Am J Physiol Renal Physiol ; 310(9): F895-908, 2016 05 01.
Article en En | MEDLINE | ID: mdl-26887830
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Dinoprostona / Diacilglicerol Quinasa / Endotelio / Ciclooxigenasa 2 / Glomerulonefritis / Glomérulos Renales Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: Am J Physiol Renal Physiol Asunto de la revista: FISIOLOGIA / NEFROLOGIA Año: 2016 Tipo del documento: Article Pais de publicación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Dinoprostona / Diacilglicerol Quinasa / Endotelio / Ciclooxigenasa 2 / Glomerulonefritis / Glomérulos Renales Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: Am J Physiol Renal Physiol Asunto de la revista: FISIOLOGIA / NEFROLOGIA Año: 2016 Tipo del documento: Article Pais de publicación: Estados Unidos