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Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells.
Yshii, Lidia M; Denadai-Souza, Alexandre; Vasconcelos, Andrea R; Avellar, Maria Christina W; Scavone, Cristoforo.
Afiliación
  • Yshii LM; Department of Pharmacology, Molecular Neuropharmacology Laboratory, Institute of Biomedical Science ICB-1, University of São Paulo, Avenida Professor Lineu Prestes, 1524, São Paulo, 05508-900, Brazil. lidiays@yahoo.com.
  • Denadai-Souza A; Department of Pharmacology, Section of Experimental Endocrinology, Federal University of São Paulo, São Paulo, Brazil. aledenadai@hotmail.com.
  • Vasconcelos AR; Department of Pharmacology, Molecular Neuropharmacology Laboratory, Institute of Biomedical Science ICB-1, University of São Paulo, Avenida Professor Lineu Prestes, 1524, São Paulo, 05508-900, Brazil. andrea.vasconcelos@gmail.com.
  • Avellar MC; Department of Pharmacology, Section of Experimental Endocrinology, Federal University of São Paulo, São Paulo, Brazil. mcwavellar@gmail.com.
  • Scavone C; Department of Pharmacology, Molecular Neuropharmacology Laboratory, Institute of Biomedical Science ICB-1, University of São Paulo, Avenida Professor Lineu Prestes, 1524, São Paulo, 05508-900, Brazil. cristoforo.scavone@gmail.com.
J Neuroinflammation ; 12: 193, 2015 Oct 26.
Article en En | MEDLINE | ID: mdl-26502720

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neuroglía / Muerte Celular / Medios de Cultivo Condicionados / Alfa-Sinucleína Límite: Animals / Humans Idioma: En Revista: J Neuroinflammation Asunto de la revista: NEUROLOGIA Año: 2015 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neuroglía / Muerte Celular / Medios de Cultivo Condicionados / Alfa-Sinucleína Límite: Animals / Humans Idioma: En Revista: J Neuroinflammation Asunto de la revista: NEUROLOGIA Año: 2015 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Reino Unido