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The impact of MTHFR 677 C/T genotypes on folate status markers: a meta-analysis of folic acid intervention studies.
Colson, Natalie J; Naug, Helen L; Nikbakht, Elham; Zhang, Ping; McCormack, Joanna.
Afiliación
  • Colson NJ; School of Medical Science, Griffith University, Gold Coast, Australia. n.colson@griffith.edu.au.
  • Naug HL; Menzies Health Institute Queensland, Gold Coast, Australia. n.colson@griffith.edu.au.
  • Nikbakht E; School of Medical Science, Griffith University, Gold Coast, Australia.
  • Zhang P; Menzies Health Institute Queensland, Gold Coast, Australia.
  • McCormack J; School of Medical Science, Griffith University, Gold Coast, Australia.
Eur J Nutr ; 56(1): 247-260, 2017 Feb.
Article en En | MEDLINE | ID: mdl-26497154
PURPOSE: Methylenetetrahydrofolate reductase (MTHFR) is a key folate pathway enzyme with the T variant of the MTHFR gene increasing the risk of low folate status, particularly coupled with low folate intake. As genetic variability of MTHFR influences folate status, it is important to ensure an adequate intake that overrides genetic effects but minimises any adverse effects. Our aim was to assess the influence of MTHFR genotype on folate status followed by response to supplementation. METHODS: We performed a meta-analysis of ten folate intervention studies to assess the degree to which MTHFR C677T genotype influenced plasma homocysteine and serum folate levels as measures of folate status. We then examined response after supplementation at intake values up to the upper tolerable limit. RESULTS: The MTHFR 677TT genotype was associated with higher plasma homocysteine (2.7 µmol/L, TT vs. CT/CC; 2.8 µmol/L, TT vs. CC) and lower serum folate (2.5 nmol/L, TT vs. CT/CC; 3.6 nmol/L, TT vs. CC). In two studies, the TT groups had mean plasma Hcy >15 µmol/L. Serum folate levels were >7 nmol/L for all genotype groups. After supplementation of 400 up to 1670 µg DFEs of folic acid or folic acid + fortified foods and/or natural food folates for a minimum of 4 weeks, there were no significant differences in plasma homocysteine levels; however, individuals with the TT genotype had a lower serum folate response to supplementation (7.2 nmol/L, TT vs. CT/CC; 8.7 nmol/L, TT vs. CC). CONCLUSIONS: This meta-analysis confirms observations from observational and intervention studies that MTHFR TT genotype is associated with increased plasma homocysteine and lowered serum folate and less response to short-term supplementation. The results can be used for modelling and guiding personalised intake recommendations for the nutrient folate.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Biomarcadores / Estado Nutricional / Metilenotetrahidrofolato Reductasa (NADPH2) / Ácido Fólico / Genotipo Tipo de estudio: Clinical_trials / Observational_studies / Systematic_reviews Límite: Humans Idioma: En Revista: Eur J Nutr Asunto de la revista: CIENCIAS DA NUTRICAO Año: 2017 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Alemania

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Biomarcadores / Estado Nutricional / Metilenotetrahidrofolato Reductasa (NADPH2) / Ácido Fólico / Genotipo Tipo de estudio: Clinical_trials / Observational_studies / Systematic_reviews Límite: Humans Idioma: En Revista: Eur J Nutr Asunto de la revista: CIENCIAS DA NUTRICAO Año: 2017 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Alemania