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Expression and therapeutic targeting of dopamine receptor-1 (D1R) in breast cancer.
Borcherding, D C; Tong, W; Hugo, E R; Barnard, D F; Fox, S; LaSance, K; Shaughnessy, E; Ben-Jonathan, N.
Afiliación
  • Borcherding DC; Department of Cancer Biology, University of Cincinnati, Cincinnati, OH, USA.
  • Tong W; Department of Pathology, University of Cincinnati, Cincinnati, OH, USA.
  • Hugo ER; Department of Cancer Biology, University of Cincinnati, Cincinnati, OH, USA.
  • Barnard DF; Department of Cancer Biology, University of Cincinnati, Cincinnati, OH, USA.
  • Fox S; Department of Cancer Biology, University of Cincinnati, Cincinnati, OH, USA.
  • LaSance K; Department of Radiology, University of Cincinnati, Cincinnati, OH, USA.
  • Shaughnessy E; Department of Surgery, University of Cincinnati, Cincinnati, OH, USA.
  • Ben-Jonathan N; Department of Cancer Biology, University of Cincinnati, Cincinnati, OH, USA.
Oncogene ; 35(24): 3103-13, 2016 06 16.
Article en En | MEDLINE | ID: mdl-26477316
Patients with advanced breast cancer often fail to respond to treatment, creating a need to develop novel biomarkers and effective therapeutics. Dopamine (DA) is a catecholamine that binds to five G protein-coupled receptors. We discovered expression of DA type-1 receptors (D1Rs) in breast cancer, thereby identifying these receptors as novel therapeutic targets in this disease. Strong to moderate immunoreactive D1R expression was found in 30% of 751 primary breast carcinomas, and was associated with larger tumors, higher tumor grades, node metastasis and shorter patient survival. DA and D1R agonists, signaling through the cGMP/protein kinase G (PKG) pathway, suppressed cell viability, inhibited invasion and induced apoptosis in multiple breast cancer cell lines. Fenoldopam, a peripheral D1R agonist that does not penetrate the brain, dramatically suppressed tumor growth in two mouse models with D1R-expressing xenografts by increasing both necrosis and apoptosis. D1R-expressing primary tumors and metastases in mice were detected by fluorescence imaging. In conclusion, D1R overexpression is associated with advanced breast cancer and poor prognosis. Activation of the D1R/cGMP/PKG pathway induces apoptosis in vitro and causes tumor shrinkage in vivo. Fenoldopam, which is FDA (Food and Drug Administration) approved to treat renal hypertension, could be repurposed as a novel therapeutic agent for patients with D1R-expressing tumors.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Receptores Dopaminérgicos / Antagonistas de Dopamina / Agonistas de Dopamina Límite: Animals / Female / Humans / Middle aged Idioma: En Revista: Oncogene Asunto de la revista: BIOLOGIA MOLECULAR / NEOPLASIAS Año: 2016 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Receptores Dopaminérgicos / Antagonistas de Dopamina / Agonistas de Dopamina Límite: Animals / Female / Humans / Middle aged Idioma: En Revista: Oncogene Asunto de la revista: BIOLOGIA MOLECULAR / NEOPLASIAS Año: 2016 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido