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Impaired light detection of the circadian clock in a zebrafish melanoma model.
Hamilton, Noémie; Diaz-de-Cerio, Natalia; Whitmore, David.
Afiliación
  • Hamilton N; a Center for Cell and Molecular Dynamics; Department of Cell and Development Biology ; University College London ; London , UK.
Cell Cycle ; 14(8): 1232-41, 2015.
Article en En | MEDLINE | ID: mdl-25832911
The circadian clock controls the timing of the cell cycle in healthy tissues and clock disruption is known to increase tumourigenesis. Melanoma is one of the most rapidly increasing forms of cancer and the precise molecular circadian changes that occur in a melanoma tumor are unknown. Using a melanoma zebrafish model, we have explored the molecular changes that occur to the circadian clock within tumors. We have found disruptions in melanoma clock gene expression due to a major impairment to the light input pathway, with a parallel loss of light-dependent activation of DNA repair genes. Furthermore, the timing of mitosis in tumors is perturbed, as well as the regulation of certain key cell cycle regulators, such that cells divide arhythmically. The inability to co-ordinate DNA damage repair and cell division is likely to promote further tumourigenesis and accelerate melanoma development.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Pez Cebra / Relojes Circadianos / Luz / Melanoma Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals Idioma: En Revista: Cell Cycle Año: 2015 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Pez Cebra / Relojes Circadianos / Luz / Melanoma Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals Idioma: En Revista: Cell Cycle Año: 2015 Tipo del documento: Article Pais de publicación: Estados Unidos