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FBXO4 loss facilitates carcinogen induced papilloma development in mice.
Lian, Zhaorui; Lee, Eric K; Bass, Adam J; Wong, Kwok K; Klein-Szanto, Andres Jp; Rustgi, Anil K; Diehl, J Alan.
Afiliación
  • Lian Z; a Institute for Regenerative Medicine; University of Pennsylvania School of Medicine ; Philadelphia , PA , USA.
Cancer Biol Ther ; 16(5): 750-5, 2015.
Article en En | MEDLINE | ID: mdl-25801820
Cyclin D1 is frequently overexpressed in esophageal squamous cell carcinoma (ESCC) and is considered a key driver of this disease. Mutations in FBXO4, F-box specificity factor that directs SCF-mediated ubiquitylation of cyclin D1, occur in ESCC with concurrent overexpression of cyclin D1 suggesting a potential tumor suppressor role for FBXO4. To evaluate the contribution of FBXO4-dependent regulation cyclin D1 in esophageal squamous cell homeostasis, we exposed FBXO4 knockout mice to N-nitrosomethylbenzylamine (NMBA), an esophageal carcinogen. Our results revealed that loss of FBXO4 function facilitates NMBA induced papillomas in FBXO4 het (+/-) and null (-/-) mice both by numbers and sizes 11 months after single dose NMBA treatment at 2mg/kg by gavage when compared to that in wt (+/+) mice (P < 0.01). No significant difference was noted between heterozygous or nullizygous mice consistent with previous work. To assess cyclin D1/CDK4 dependence, mice were treated with the CDK4/6 specific inhibitor, PD0332991, for 4 weeks. PD0332991 treatment (150mg/kg daily), reduced tumor size and tumor number. Collectively, our data support a role for FBXO4 as a suppressor of esophageal tumorigenesis.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias Esofágicas / Carcinoma de Células Escamosas / Ciclina D1 / Proteínas F-Box Límite: Animals / Humans Idioma: En Revista: Cancer Biol Ther Asunto de la revista: NEOPLASIAS / TERAPEUTICA Año: 2015 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias Esofágicas / Carcinoma de Células Escamosas / Ciclina D1 / Proteínas F-Box Límite: Animals / Humans Idioma: En Revista: Cancer Biol Ther Asunto de la revista: NEOPLASIAS / TERAPEUTICA Año: 2015 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos