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Effects of whole cigarette smoke on human beta defensins expression and secretion by oral mucosal epithelial cells.
Wang, Wen-Mei; Ye, Pei; Qian, Ya-Jie; Gao, Ya-Fan; Li, Jing-Jing; Sun, Fang-Fang; Zhang, Wei-Yun; Wang, Xiang.
Afiliación
  • Wang WM; Department of Oral Medicine, Institute and Hospital of Stomatology, Nanjing University Medical School, 30 Zhongyang Road, Nanjing, 210008 China.
  • Ye P; Department of Oral Medicine, Institute and Hospital of Stomatology, Nanjing University Medical School, 30 Zhongyang Road, Nanjing, 210008 China.
  • Qian YJ; Department of Oral Medicine, Institute and Hospital of Stomatology, Nanjing University Medical School, 30 Zhongyang Road, Nanjing, 210008 China.
  • Gao YF; Department of Oral Medicine, Institute and Hospital of Stomatology, Nanjing University Medical School, 30 Zhongyang Road, Nanjing, 210008 China.
  • Li JJ; Department of Oral Medicine, Institute and Hospital of Stomatology, Nanjing University Medical School, 30 Zhongyang Road, Nanjing, 210008 China.
  • Sun FF; Department of Prosthodontics, Institute and Hospital of Stomatology, Nanjing University Medical School, Nanjing, 21008 China.
  • Zhang WY; Immunology and Reproduction Biology Laboratory, Medical School, Nanjing University, 22 Hankou Road, Nanjing, 210093 China.
  • Wang X; Immunology and Reproduction Biology Laboratory, Medical School, Nanjing University, 22 Hankou Road, Nanjing, 210093 China.
Tob Induc Dis ; 13(1): 3, 2015.
Article en En | MEDLINE | ID: mdl-25635179
BACKGROUND: Cigarette smoke a recognized risk factor for many systemic diseases and also oral diseases. Human beta defensins (HBDs), a group of important antimicrobial peptides expressed by the epithelium, are crucial for local defense and tissue homeostasis of oral cavity. The aim of this study was to evaluate potential effects of whole cigarette smoke (WCS) exposure on the expression and secretion of HBDs by oral mucosal epithelial cells. METHODS: Immortalized human oral mucosal epithelial (Leuk-1) cells were exposed to WCS for various time periods. HBD-1, -2 and -3 expression and subcellular localization were detected by real time qPCR, immunofluorescence assay and confocal microscopy. According to the relative fluorescent intensity, the expression levels of HBD-1, -2 and -3 were evaluated by digital image analysis system. The alteration of HBD-1, -2 and -3 secretion levels was measured by the Enzyme-Linked Immunosorbent Assay. RESULTS: WCS exposure remarkably attenuated HBD-1 expression and secretion while clearly enhanced HBD-2, -3 expression levels and HBD-2 secretion by Leuk-l cells. It appeared that there was no significant effect of WCS exposure on HBD-3 secretion. CONCLUSIONS: WCS exposure could modulate expression and secretion of HBDs by oral mucosal epithelial cells, establishing a link between cigarette smoke and abnormal levels of antimicrobial peptides. The present results may give a new perspective to investigate smoking-related local defense suppression and oral disease occurrence.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Tob Induc Dis Año: 2015 Tipo del documento: Article Pais de publicación: Grecia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Tob Induc Dis Año: 2015 Tipo del documento: Article Pais de publicación: Grecia