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Cytotoxic T lymphocytes and natural killer cells display impaired cytotoxic functions and reduced activation in patients with alcoholic hepatitis.
Støy, Sidsel; Dige, Anders; Sandahl, Thomas Damgaard; Laursen, Tea Lund; Buus, Christian; Hokland, Marianne; Vilstrup, Hendrik.
Afiliación
  • Støy S; Department of Hepatology and Gastroenterology, Aarhus University Hospital, Aarhus, Denmark; and sidsel.stoy@post.au.dk.
  • Dige A; Department of Hepatology and Gastroenterology, Aarhus University Hospital, Aarhus, Denmark; and.
  • Sandahl TD; Department of Hepatology and Gastroenterology, Aarhus University Hospital, Aarhus, Denmark; and.
  • Laursen TL; Department of Hepatology and Gastroenterology, Aarhus University Hospital, Aarhus, Denmark; and.
  • Buus C; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
  • Hokland M; Department of Biomedicine, Aarhus University, Aarhus, Denmark.
  • Vilstrup H; Department of Hepatology and Gastroenterology, Aarhus University Hospital, Aarhus, Denmark; and.
Am J Physiol Gastrointest Liver Physiol ; 308(4): G269-76, 2015 Feb 15.
Article en En | MEDLINE | ID: mdl-25501547
The dynamics and role of cytotoxic T lymphocytes (CTLs), natural killer (NK) cells, and NKT cells in the life-threatening inflammatory disease alcoholic hepatitis is largely unknown. These cells directly kill infected and damaged cells through, e.g., degranulation and interferon-γ (IFNγ) production, but cause tissue damage if overactivated. They also assist tissue repair via IL-22 production. We, therefore, aimed to investigate the frequency, functionality, and activation state of such cells in alcoholic hepatitis. We analyzed blood samples from 24 severe alcoholic hepatitis patients followed for 30 days after diagnosis. Ten healthy abstinent volunteers and 10 stable abstinent alcoholic cirrhosis patients were controls. Using flow cytometry we assessed cell frequencies, NK cell degranulation capacity following K562 cell stimulation, activation by natural killer group 2 D (NKG2D) expression, and IL-22 and IFNγ production. In alcoholic hepatitis we found a decreased frequency of CTLs compared with healthy controls (P < 0.001) and a similar trend for NK cells (P = 0.089). The NK cell degranulation capacity was reduced by 25% compared with healthy controls (P = 0.02) and by 50% compared with cirrhosis patients (P = 0.04). Accordingly, the NKG2D receptor expression was markedly decreased on NK cells, CTLs, and NKT cells (P < 0.05, all). The frequencies of IL-22-producing CTLs and NK cells were doubled compared with healthy controls (P < 0.05, all) but not different from cirrhosis patients. This exploratory study for the first time showed impaired cellular cytotoxicity and activation in alcoholic hepatitis. This is unlikely to cause hepatocyte death but may contribute toward the severe immune incompetence. The results warrant detailed and mechanistic studies.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Células Asesinas Naturales / Activación de Linfocitos / Linfocitos T Citotóxicos / Citotoxicidad Inmunológica / Hepatitis Alcohólica / Inmunidad Celular Tipo de estudio: Diagnostic_studies / Observational_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Am J Physiol Gastrointest Liver Physiol Asunto de la revista: FISIOLOGIA / GASTROENTEROLOGIA Año: 2015 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Células Asesinas Naturales / Activación de Linfocitos / Linfocitos T Citotóxicos / Citotoxicidad Inmunológica / Hepatitis Alcohólica / Inmunidad Celular Tipo de estudio: Diagnostic_studies / Observational_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Am J Physiol Gastrointest Liver Physiol Asunto de la revista: FISIOLOGIA / GASTROENTEROLOGIA Año: 2015 Tipo del documento: Article Pais de publicación: Estados Unidos