DNA end resection is needed for the repair of complex lesions in G1-phase human cells.
Cell Cycle
; 13(16): 2509-16, 2014.
Article
en En
| MEDLINE
| ID: mdl-25486192
Palabras clave
ATM, Ataxia telangiectasia mutated; ATR, Ataxia telangiectasia and Rad3-related; BLM, Bloom syndrome protein; BRCA1, breast cancer 1, early onset; CENP-F, centromere protein F; CtIP; CtIP, CTBP-interacting protein; DAPI, 4',6-diamidino-2-phenylindole; DSB, double strand break; EXO1; EXO1, exonuclease 1; FCS, fetal calf serum; HR, homologous recombination; IR, ionizing radiation; LET, linear energy transfer; MEF, mouse embryonic fibroblasts; MMEJ, microhomology-mediated end joining; MRE11; MRE11, meiotic recombination 11 homolog A; NHEJ, none homologous end joining; PARP, poly (ADP-ribose) polymerase; RAD51, DNA repair protein RAD51 homolog 1; RPA, replication protein A; WRN, Werner syndrome; complex DNA damage; double-strand break repair; kd, knockdown; resection in G1-phase; siRNA, small interfering RNA; ssDNA, single stranded DNA; wt, wild-type
Texto completo:
1
Colección:
01-internacional
Base de datos:
MEDLINE
Asunto principal:
Fase G1
/
Roturas del ADN de Doble Cadena
/
Reparación del ADN por Unión de Extremidades
Límite:
Humans
Idioma:
En
Revista:
Cell Cycle
Año:
2014
Tipo del documento:
Article
País de afiliación:
Alemania
Pais de publicación:
Estados Unidos