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Thrombotic safety of prothrombin complex concentrate (Beriplex P/N) for dabigatran reversal in a rabbit model.
Herzog, Eva; Kaspereit, Franz J; Krege, Wilfried; Doerr, Baerbel; van Ryn, Joanne; Dickneite, Gerhard; Pragst, Ingo.
Afiliación
  • Herzog E; CSL Behring GmbH, Marburg, Germany. Electronic address: Eva.Herzog@cslbehring.com.
  • Kaspereit FJ; CSL Behring GmbH, Marburg, Germany.
  • Krege W; CSL Behring GmbH, Marburg, Germany.
  • Doerr B; CSL Behring GmbH, Marburg, Germany.
  • van Ryn J; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
  • Dickneite G; CSL Behring GmbH, Marburg, Germany.
  • Pragst I; CSL Behring GmbH, Marburg, Germany.
Thromb Res ; 134(3): 729-36, 2014 Sep.
Article en En | MEDLINE | ID: mdl-25084749
INTRODUCTION: In vivo animal data have shown prothrombin complex concentrate (PCC) to be effective in preventing bleeding induced by excessive plasma levels of the direct thrombin inhibitor dabigatran. This animal model study was designed to determine the risk of thrombosis associated with administration of a PCC (Beriplex P/N) to reverse dabigatran-induced bleeding. MATERIALS AND METHODS: Anesthetized rabbits were treated with initial 0, 75, 200 or 450 µg kg(-1) dabigatran boluses followed by continuous infusions to maintain elevated plasma dabigatran levels. At 15 min after the start of dabigatran administration, PCC doses of 0, 50 or 300 IU kg(-1) were administered. Thereafter, coagulation in an arteriovenous (AV) shunt was evaluated and histopathologic examination for thrombotic changes performed. Venous thrombosis was also assessed in a modified Wessler model. RESULTS: At the suprapharmacologic dose of 300 IU kg(-1), PCC increased thrombus weight during AV shunting, but this effect could be prevented by dabigatran at all tested doses. AV shunt occlusion after PCC administration was delayed by 75 µg kg(-1) dabigatran and abolished by progressively higher dabigatran doses. High-dose treatment with 300 IU kg(-1) PCC resulted in histologically evident low-grade pulmonary thrombi; however, that effect could be blocked by dabigatran in a dose-dependent manner (p=0.034). In rabbits treated with high-dose PCC, dabigatran inhibited thrombus formation during venous stasis. PCC effectively reversed dabigatran-induced bleeding. CONCLUSIONS: In this animal study, thrombosis after PCC administration could be prevented in the presence of dabigatran. PCC reversed dabigatran-induced excessive bleeding while retaining protective anticoagulatory activity of dabigatran.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Coagulación Sanguínea / Factor IX / Factor VII / Factor X / Protrombina / Hemostáticos / Trombosis de la Vena / Dabigatrán / Hemorragia Tipo de estudio: Etiology_studies / Prognostic_studies / Risk_factors_studies Límite: Animals Idioma: En Revista: Thromb Res Año: 2014 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Coagulación Sanguínea / Factor IX / Factor VII / Factor X / Protrombina / Hemostáticos / Trombosis de la Vena / Dabigatrán / Hemorragia Tipo de estudio: Etiology_studies / Prognostic_studies / Risk_factors_studies Límite: Animals Idioma: En Revista: Thromb Res Año: 2014 Tipo del documento: Article Pais de publicación: Estados Unidos